Tue, Jun 06, 2023 7:59PM • 2:52:02
SUMMARY KEYWORDS
inhibit, drug, inflammation, good, npk, cancer, activate, add, stimulate, disease, lead, mtor, work, helps, mast cells, fibrosis, patient, manage, ox, patel
SPEAKERS
Bill Clearfield
02:00
Hello everyone. Hi Dr. alasa
02:04
Hey how are you?
02:05
Excellent.
02:06
I didn’t, I just came up from the shower refreshing. Ah, yeah. Today we’re gonna talk about a very interesting topic. I think it’s gonna be entertaining. I even invited Dr. Class to attend it and it’s very important topic as well. Let me just find where it is where I put it Ah okay. Okay, okay, okay, so this is
02:54
John, yo,
02:57
um, I’m actually on a, you know, I’m out today so can you I put made you to co host
03:04
Okay, now.
03:05
Are you leaving me? I’m gonna try and listen, but I really can’t be running the show here tonight. So,
03:13
okay, now we need your feedback and your all the good stuff that you can add.
Bill Clearfield 03:19
Well, I’m I’m visiting grandkids and it’s my last night with them. So I right. You’re gonna, you’re gonna have to carry on without my hair. You can do it. You guys can do it.
03:33
Yeah, John. was able to do it. Come on.
03:37
I’m just clicking the admit button.
03:40
Okay.
03:42
That’s all okay, so I think I’ll fill the slide now
03:55
yes, great, beautiful. Oh, can I and then you want to wait for people to come
04:05
some time. If it’s after eight o’clock your time You can want us to just get started.
04:12
Okay, how much people we have here.
04:15
I got 21 so far. Oh, that’s
04:17
a lot. Okay, that’s good.
04:20
We’ve gotten 10 In the last row,
04:23
there is molecule that speed hidden or long time can you give me so I can record it let’s do this. Like can we can you get me the money what’s the name of it? The cool house yeah, I’ll do that. So the recording it there. But I don’t know if I can record it if you recorded yourself there. I think I can. Okay, so you’re recording it? That’s fine, you know can get it from you. So anyway. Yeah, I’m Dr. Melissa, the founder of chronic disease prevention and management. And we are looking for any integration and any trend that we can use it to redefine medicine. Dr. Joseph James is the one who brought this molecule. When we are talking about oxytocin and Benadryl, what he said well, there is a molecule that that stabilizes the mast cell. And it has many other many other qualities and and it’s been used in Japan and it’s FDA approved for for oral answers. That’s it. And then in certain way, it’s been discontinued in the United States for no reason. Except they say they find better medication for mouth ulcers. But then there’s a lot of sophistication happened with this drug and now there’s form of oral tablets. It’s been dispensed here and compounding pharmacy. Doctor might be Meramec pharmacy has carried those this this drug in the form of 40 milligram dose. And it has many many good healing effects, because it has many targets and critical targets in and our biochemistry and it helps to restore a lot of, of the pathological Doctor physiology by targeting those critical enzymes in our body. And as you see, it’s a new trend of medicine, I think, and it has the potential to manage and present chronic disease across all medical specialities. That’s how big this molecule that’s when you can use it for degenerative disease, autoimmune disease, allergies, cancer, and you’ll be surprised why that this drug can be used for all this chronic inflammation, degenerative autoimmune cancer and also genetic diseases. And that that’s the that’s the beauty of this drug that other drugs that we know that they don’t really touch anything genetic, but this drug does and the way the does help in managing genetic diseases is that those 30% of genetic diseases because of mutation, we call them nonsense mutation where we have abnormal stop codons and the reading of the DNA of the genes and those messenger RNA when they have to stop codons. And those stop codons. I think there are three of them. And when I was teaching in college, I was trying to simplify it for the students. And the name of those three, it’s your garden, you go away and you are away. And that is that’s those are abbreviations for for you are gone you a G you go away UGA and you are away you a so those are stop codons where they don’t have any cannot be translated into any amino acids. And it happened that the genes of those 30% of genetic diseases, they have this nonsense mutation that stop codons in the middle of that messenger RNA of that specific protein. And so when that DNA is being prescribed, transcribed into messenger RNA you will see that stuff going on the middle, and there’s enzyme and it’s called NMD. And it’s nonsense. messenger RNA mediated decay, which is an enzyme that binds those nonsense codon, and those messengers have RNA, and they just destroy them. So those messenger RNA will not be translated into a protein and you end up with of a missing protein that leads to that specific genetic disease. Let’s say one of the example is Cystic Fibrosis, right? And we know that the missing genes is the coloration channel, genes that that produce the protein for the chloric chloride channels, right. And what happened is that we can treat cystic fibrosis is a genetic disease by giving I’m not I’m not Synnex because it will inhibit this NMD enzyme that breaks down those messenger RNA that has the stop codons and doing that I can produce again those missing protein, and I can reverse the pathology of cystic fibrosis. This is just an example but almost 80% of genetic disease, there are nonsense, and that is going to be another talk. I think it’s going to be the Session Two of em luxon ox, session one we’re going to talk about how I’m not an ox, modulate the immune system by blocking enzymes, which is the it’s called the NFA beta or NF kappa B and NF kappa B is an enzyme that that express the pro oxidant enzyme like Knox enzyme and generate the free radicals. And generally the free radicals is behind all that degeneration. And inflammation. So think about this enzyme was stopped put the fire down and neutralize out in other words, stop the production of those excessive of those free radicals means that all the inflammation will go down. Whether this inflammation is th one mediated th two mediated or th 17 mediated because we know that there are three kinds of inflammation. So because of that, we can use it to manage almost any chronic inflammation, any neurodegenerative disease, any autoimmune disease, including allergies, because it’s also mast cell mast cell activation, on other words, it stabilized the mast cells so it’s an activate the mast cells. And definitely this drug can be used in all kinds of inflammation, whether it’s th one or th th two or TF 17 dominant inflammation you know th two it’s the allergic Teal Swan is cytotoxic T cells mediated autoimmune diseases, like systemic lupus or like multiple sclerosis or rheumatoid arthritis or it could be th 17 mediated like for ISIS. All of these can be inhibited by this. I’m lucky to not by blocking the NfkB veto. So it definitely can be used to manage also cancer because cancer can also be due to nonsense mutation of the genes that leads to turning those cells into into cancer. So there’s many cancers that can be treated with maxing out and there’s lots of studies about them. And that will be the question number two of em LuxLock. So that’s the view about the threat, I don’t think since I studied medicine, and I think everybody here did not hear about a drug that helps to increase the expression of mutated genes of the medical doctor and I think all of us in the same so this is kind of a new door as we’ve opened, that we have some solutions for those hard to treat cases, especially those genetic disease, including cancer, we can reverse the abnormal mutation that’s happening that is the root cause of turning those cells into cancer by by by inhibition of NMD enzyme and by little by reversing the mutation back to normal where they can express that missing protein that leads to the whole pathology. And so because of its quality, and mechanism of action, inhibiting NF gay betta, inhibiting the mTOR and you can see an apron they talk a lot about mTOR. Although mTOR is not really the upstream enzyme or genes. The reality is that a NPK it’s more important than I’m poor. I don’t know why. And a for me to entertain I’m sure a lot mTOR is actually inhibited by NPK and mTOR. When they are stimulated, they stimulate stem cell differentiation, proliferation but also still stimulate the cancer as well. So to stimulate the growth and inhibiting mTOR needs to accomplish and so it’s very important when you are treating
14:23
cancer or treating inflammatory diseases, you inhibit the mTOR to stimulate the autophagy. And what’s controlling them for is the ANP k. So I have BK it’s very important enzyme. It’s a kinase enzyme that that turns the enzymes are turned enzymes off and on by adding phosphate group to it. That’s how they they they work. It’s the kinase right so versus phosphatase removing the phosphate. This is adding the phosphate and I am MBK we’re going to talk about it. It’s a very critical enzyme that mediates almost all the action like fasting and exercise. It works through activation of MMP and when you once you activate their amtk you end up increasing the mitochondrial function and biogenesis and you increase the glucose uptake which is insulin sensitivity and you end up with weight loss. You end up with like lysis beta oxidation of fat which means that by activation ANP k you can manage all metabolic syndrome. We know the metabolic syndrome, right the way the weight gain arthrosclerosis diabetes, the hypertension right and just by activating that make NPK we can manage that metabolic syndrome. Now I said that this molecule works like steroid but without side effects of steroids and that’s very important statement. I need everybody to understand that as a doctor. So I’m not Synnex does inhibit the clocks and locks enzyme and doesn’t have it then fkb But what it does it stimulate dmpk. Now steroids, it does the same thing. They inhibit NfkB B which means they put down the free radicals right the KNOX enzymes expression will go down and their activities so you don’t have much of free radicals. The inflammation will go down the marks and blocks will go down right you don’t have those prostaglandins and all those leukotrienes and things that that you know, turn the inflammation on. So this is steroids and I’m Luxottica does the same thing right but what’s the difference here is the critical important point is that steroids inhibit the MMA NPK and that leads to metabolic syndrome, while I’m lucky enough to activate bae and PK. So that’s the difference. And it’s really important to know that so that’s reason why steroid is good for acute conditions, but when he uses chronically you end up with oxidative stress because it inhibit the NPK which is mean if you want to use the Stewart’s will be good if you add polyphenols I’m not saying ox which helps to activate the a MP can reverse the side effects of cortisol but we find out that I’m not saying X can replace steroids and almost every application of steroids especially when it comes into Quranic right. I’m not saying that the dumping effect of suppressing effect of the steroid is way higher than I’m Luxor Knox, but still get Tempe replaced because it does suppress the same enzyme that steroids does, but not in a severe way where it will lead to atrophy. But enough to manage those conditions but without the side effects of steroids, which is blocking the ANP. Okay, so those terminology of a NPK and a Kappa Beta. Those are very important. mTOR which, you know, April they like to talk those doctors have a friend they like talk about it. It’s downstream. Yes, it’s important to inhibit them. If you want to use a toffee G but at the same time we need to stimulate them if we want to stimulate the stem cells, right. But if you want to treat cancer, you need to inhibit mTOR. But you can argue that inhibiting mTOR is very important for anti aging because this will lead to autophagy of the senescence cells and inflammasomes. And we know the senescence cells inflammasome. They secrete interleukin six which inhibit the stem cell proliferation. Let me just close the door here. Okay. I can’t start planning. Alright, so I think I just gave a very big introduction about I’m not Synnex I’m trying to break it down, simplify it in and before we go through all the slides, so you can be prepared that this is very important molecule and and it does gives us an insight about other farmers, other other other pharmaceutical products like steroids and, and other drugs, right. So I’m not an Oxford small molecule which has been used in treatment of atopic condition, and I think they using it for for oral ulcers of this and previously, but it’s not currently available United States did they discontinue it because they say well, there’s better drug for Afters also for those oral oral ulcers. But in Japan, they continue, they continue using it and then they develop from drugs for asthma. And they have clinical trials for it which is very successful and the safety of the drug. It’s very, very phenomenal. I mean it’s really good um, not select has also been legally utilized and other western Japan as a treatment for asthma because it’s a mast cell activation, it’s the mast cell stabilizer and inhibit inflammation right, inhibit the th two inflammation and th 17 And those two inflammation if 17 th two are mediating the asthma and also it’s been used for chronic pulmonary diseases characterized by inflammation of the lower spine three tracks right you can use it for even obstructive diseases COPD, chronic obstructive pulmonary diseases, because it does help to suppress the inflammation thing like cortisone without the side effect of the steroids. That’s the beauty. Why because as I said, it’s activate the ANP game. So immune, so I’m not synopsis immune modulators. I don’t want to say it’s immune suppression because the users of those drugs does not have risk of having infection and cancer like steroid does. So steroid does suppress the immune system does suppress the th one too much to the point it compromised immune system um luxon ox does damage the immune system. So it’s an immune modulator effect, and has been subject of study which has purpose in the drug for potential therapeutic potential and metabolic and inflammatory disease so I can use it for weight loss for diabetes or hypertension because it activate the am PK so it’s good for metabolic and also good for inflammatory because it inhibits the NF K Kappa Beta right okay and FK kappa NF kappa B. And doing that I can use it for treating inflammatory disease but in addition, it doesn’t have the Coxon locks enzyme and Knox enzyme as well, like the steroid so this is the molecule how they look like. And this molecule is been FDA approved for oral ulcers right. And now they find better drug. You can use methylene blue for that.
22:17
So that clinical application of MX Linux because I’m not saying ox and habit that tank binding kinase one which is the GTB k one and the TB k one and also to inhibit the nuclear factor kappa B. And both of those kinases inhibit the NFK B, right. What’s important is the NFA B is inhibited by those Tbk one and IK k there are kinase proteins right and how they inhibit they are the postepay to the other enzyme, and that enzyme will turn off. So they are kinase proteins and they have many names but eventually they inhibit the NF K beta or NF kappa B. That’s important enzyme right? Several studies that demonstrate that the usefulness throw it’s evidence down regulation of the immune system. Once you have the NfkB you decrease the activity of the KNOX enzyme and you suppress the production of the free radicals. And when you do that, the macrophage will will match your maturation into m one or m two arms 17 will be suppressed because in order for polarization of macrophages and lymphocytes, right, and turn them into active they need to have free radicals. And if you have too much of inflammation and too much of active macrophages and too much of DF 17 or r th two or th one all of this will be suppressed just by decreasing the free radicals inside them. And also those in Zion, which is the NF K betta are responsible and NF kappa B are responsible for expression and stimulation are the free radicals of all of the cytokines whether those cytokines are for innate immunity interleukin one, or interleukin six or interleukin four or interferon gamma, all those cytokines expression depend on activation of NF KB. So if you inhibit that you inhibit this you suppress the whole immune system. I like to say dumping the immune system but not suppressing it to the point it’s compromised. The immune system to fight to compromise immune system against cancer against infection. So that’s a very important point that we need to learn from here. And so I can use it for for all kinds of inflammatory conditions including asthma including rheumatoid arthritis, multiple sclerosis, systemic sclerosis. What are all those inflammation all this neurodegenerative diseases I can use it for me typo like or for weight loss for diabetes or hypertension. And so, if you see here the how the M luxon ox blocks the Tbk one which is a kinase enzyme, right? And how this will of course this kinase is different I think than fk be right see here. And so if you inhibit the T V 1k, why’d you inhibit the NF K B? And if you’d have done F, kappa B, you inhibit the cytokines expression, the free radicals, the NOx enzyme, the free radicals and free radicals are very important to to stimulate the expression of those inflammatory cytokines, and those inflammatory cytokines including to neocon one, interleukin six interleukin four name and all those can be suppressed, or damped by inhibition of NfkB. So I’m lucky to eventually inhibit the NfkB throw inhibition of Tbk one, and they are just kinase protein, how they do that they add the phosphate group to the NfkB and that turns it on. Versus phosphatase enzyme they remove the phosphatase phosphate group and they then the NF KB will be activated. Okay. And if you see here, let’s say this is a macrophage, right, and this macrophage how they get activated. Let’s say they’re activated by bacteria, lipid polysaccharides, right. So, macrophage is the lack of polysaccharides. This is the toll like receptor in Alltel acceptor for they they taste that lipid polysaccharides and that will signal the activation of the Tbk one and then TVK will activate the NfkB directly and this will lead to expression most of the time when you have like replaced saccharides here, the macrophage will turn into M 17 and we’ll turn on the th 17 And then ts 17 will secrete those interleukin six six and an interleukin 17 which will oppose the differentiation of the white blood cells into neutral field because that’s how you kill the bacteria by natural field. But if you see here that how the toll like receptors in the macrophages is the one who will taste those bacteria and the macrophage will turn into m 17 And then that the macrophages will release those interleukin six and interleukin one and all those cytokines to eventually turn the naive T cells into th 17 And then secrete interleukin six to to differentiate the white blood cells into neutral field and neutrophil will go ahead and eat those along with this will activation also interleukin four so you will have some th to active as well to produce antibodies that that will lead to neutralizing those bacteria because you need to feel nega antibody to kill and so you need th two and Ts 17 to really attack and destroy those materials.
27:58
In addition to that, as you see here, when when we have those fat cells or when we have obese patients, and there’s too much of fat cells, those fat cells they are not good because they secrete interleukin six and they can lead to inflammation right you know when you wait day that’s not good. It’s not healthy when you see somebody with big belly. That’s that’s not good because all those fat cells they tend to secrete interleukin six. And what are the things that we can use to manage and even to burn those fat is using um Luxa Max and Max Knox will inhibit the expression of interleukin six so NfkB and also throw inhibition of phospho Dire Straits eventually, that leads to decrease of the cytokines that’s released from those fat cells and decrease the macrophage from being polarized either to M 17 Or to two and that decrease the T at 17 and PHQ. Eventually it will help to suppress the inflammation and also I’m not saying during doing that also it stimulate the life license and stimulate the beta oxidation. So it helps to decrease weight right by shrinking those fat by dissolving those bubbles of fat that’s inside those fat cells. So it does help to be more like the catabolism of fat and and led to burning fat and also these to increase the mitochondria So turning those white fat cells into brown fat cells, and that will lead to weight loss. So it’s both it’s for metabolic and also for that suppressing inflammation. So I’m not so much trade name is sofa. It’s a tri cyclic and mine carboxylic acid is a potential treatment option that was traditionally used as a treatment for recurrent aphthous ulcers. Right the mouse also enters discontinue United States. I don’t know it’s a stupid thing being done United States and 2017 why they say because they have options other options available. Although this drug has many many other applications. I mean, you can use it to manage all chronic disease across all medical specialties. You can use it for a genetic disease. 30% of them is because of the nonsense mutation. And I will have Dr. Mike beamer to go throw more about the pharmacokinetics and pharmacodynamics and about this drug and I will have him talk about the reason why they stop it. There is no really that thing that they hear about it. It’s more about well Well this has been used for off the call sir there’s another drug that can be used and seems like the big pharma the other drug it’s more patent more money. So the drag the whole thing and they forgot about this very precious molecule that is being missed. And now we learn that we can repurpose this drug and we can learn from Japanese doctors that that can be used for managing a variety of inflammatory diseases and we want to start making a game here and I just state Mike beamer is actively working on making this drug available for us. So in other countries such as Japan, I’m lucky enough to Dockerized to treat asthma allergic rhinitis, and so they also use it for conjunctivitis. So that’s mean they have a form of eyedrops not just that I mean this drug has a potential also to treat fibrotic diseases, right? So because it inhibits th 17 It inhibits interleukin six and then you have also the transforming growth. Factor Vita, the TG the transforming growth factor beta. That’s mean this product has a potential to treat liver cirrhosis and corneal ulcer saying like you remember those are 10 I dropped we talked about Yeah, this I’m not saying ox has the potential to treat fibrotic diseases. And we know fibrotic disease are one of the pandemic or epidemic diseases for hidden disease. We call it hidden epidemic disease that we fail in managing it right we fail to manage fibrotic diseases, liver cirrhosis, Corneal scar, lung fibrosis, kidney fibrosis, all this has been we’ve failed to manage we need to be able to manage adhesions, right. But this drug, along with Loeser time do have a potential to manage those fibrotic diseases because they inherit the transforming growth factor be the inflammation, which is tier 17 at mediate the fibrosis and, and they actually inhibit the fibrotic growth factor directly. And so we have potential using this drug for corneal scar for glaucoma. Because Glaucoma is a blockage in the trabeculae which is done at the angle of the of the of the anterior chamber of the eye, right? There’s fibrosis there that blocks with drainage. And we can go ahead and open that using Luxor. Right so there’s a potential to treat glaucoma, Corneal scar liver, lung fibrosis, or liver fibrosis in addition to its ability to rethrow transcriptional rethrow genes that’s been mutated because nonsense mutation and we can kind of manage 30% of genetic diseases, including cystic fibrosis, Duchenne syndrome and many others that were going to have a big, big, big another section of I’m not sure not related to managing all those nonsense mutated genes, diseases, whether it’s genetic or cancer. As we know we use it for asthma because it stabilized the mast cells or Temescal stabilizer it does not inhibit the histamine so you don’t have the sedation effect that Benadryl has when you take I’m lucky enough to just stabilize the mast cells and inhibit the inflammation and inhibit the th two to 17 which mediate inflammation of asthma. So I’m not so nice has also been shown to treat and reverse the progression of carotene liver diseases. We know why right? Because it stimulate the beta oxidation and helps to degrade those fats in the liver. And so and increase the mitochondrial function. And definitely this helped to break down the fat and so we can manage not just liver, Fatty Fatty liver diseases, but non alcoholic fatty liver disease, but also we can use it to manage liver fibrosis itself, because it didn’t have the TF 17 and transforming growth factor beta and all the growth factors, the fibrotic growth factors. Still we can use also for liver cirrhosis and we have a study I think it’s conducted by owl a he seems like a champion. He’s what utilize the mouse model and demonstrate improvement and metabolic disturbance, right metabolic syndrome, whether it took his insulin sensitivity decrease the lowering the sugar, activate the beta oxidation of the fat, it stimulate glycolysis right, and the bill of finished so you’re burning the fat, and this definitely will lead to a decrease in the hepatic steatosis ketosis. Okay. And so we can see that I’m not an ox and habit than a TV and when in heaven damn thing to be you decrease all those inflammatory cytokines and inhibit the NOx enzyme and inhibit the free radicals that will oxidize those receptors of insulin and actually it will block the reuptake of sugar it will not block that signal of insulin that leads to activation of aka t all those kinase protein which actually leads to metabolic syndrome, right, but because a que te inhibit the ANP que there’s another path here is missing. So, but it will inhibit the glucose uptake. So insulin is not good, too much of it. It will stimulate the AKA AKA T which will block the amtk and blocking the NPK will lead to insensitivity of glucose or insulin sensitivity we call them and you end up with hyperglycemia and metabolic syndrome and so inhabiting the whole process and inhabiting the AKA AKA chi aka T and inhabiting, and that will lead to stimulation of a NPK because if you have a que te you stimulate the NPK and then the asbk will increase the mitochondrial biogenesis activity or the mitochondria, it will increase the glucose and insulin sensitivity and the critical rate the reuptake and decrease the glucose level in the blood and also stimulate the lipid lysis and the beta oxidation and that leads to management of the non alcoholic fatty liver and also it leads to managing liver cirrhosis. And, and this is very important is that the unlocks do not activate the asbk by inhibiting NfkB there but also not directly can activate and stimulate dmpk We know that NPK is activated by polyphenols as well put them in ECGC Chrisitan all those stimulate the NPK.
37:57
So that is the beauty of M Lux inox decreased inflammation and it managed the metabolic syndrome steroids cannot do that. Steroids Yes, it will help the inflammation but it will it will lead to metabolic syndrome by inhibiting the NPK. And when you inhibit dmpk, you lead to oxidative stress and you lead to inflammation. That’s why you end up with atrophy and the patient gets sick from from the steroids and then you end up with this fat redistrict abdominal fat redistribution and atrophy and all the bad stuff because of the oxidative stress that’s been generated from the long term use of cortisone. So short term is good long term is not good to use cortisone because it leads to metabolic syndromes diabetic hypertension, we know that side effects common side effects. We were introduced to this but we don’t know exactly how, why, why steroids leads to secondary hypertension and diabetes. The reason because it blocks the A NPK and you can see here very clearly that how I’m looking to Knox will manage the the the the fatty liver, right and it’s doing that by inhibiting the NfkB as you see it here and it definitely doing that. It will stimulate the NPK and when you stimulate NPK you inhibit mTOR and you stimulate the autophagy. And also so you get very common Kamisama senescence cells, but you manage the whole labor by stimulation, the lipo lysis and by stimulation of beta oxidation, right mitochondria, beta oxidation and doing that this will help combat any oxidative stress and that this leads to decrease the inflammation as we learn here and it will increase that insulin sensitivity by inhibiting the NfkB Okay, so, metabolic syndrome is due to inhibition of a NPK and I’m lucky enough to activate the A and BK and we know when inhibit day and BK what would happen in deliver you will end up it will increase the lipo Genesis. If you if you have a day a BK which is syndrome you will block the beta oxidation you will COEs inhibition of the mitochondrial function. You will lead oxidative stress and you will lead to accumulation of fat in the liver and that leads to non alcoholic fatty liver. So what we need to do we need to activate the NPK and that’s what unlocked the max depth. They will activate that unlock that I mean the day when you activate the MBK you increase the mitochondrial function you increase a bit oxidation of the fat you increase the lipid lysis and and you increase the beta oxidation which means you’re burning all the fat from the liver. And definitely by doing that you manage what we call the liver fatty liver plus via inhibiting the Kissimmee teen and transforming growth factor beta. You can also manage the liver cirrhosis. Again, this just shows you unlock snacks as well as one of the drug that used to manage fatty liver but there’s other drugs that do the same thing. And they had had they helped to stimulate basically the a PK eventually okay so I’m lucky enough is a stable compound as a powder stored at 20. So you stored at minus 20 Do you want to have a shelf life of three years as a clinician of course or you know you can this is if we want to keep it for three years but for regular use, you don’t need to store it point 22 degree it had a similar chemical structure to sodium cromoglycate Because in the beginning, when they create this drug they created as a mast cell stabilizer right like sodium cromoglycate That’s our step meself analyzer and it’s topical use has been widely accepted and they’ve been using it for Afters also and its effect attributed to its anti inflammatory quality at that time. They don’t know really how it works, but now we know in detail how it works and the successful use of the treatment with favorable safety profile. So that’s the beauty here is the safety profile very important safety, safety, safety. And atopic disease as an oral agent has been established and is currently approved for treatment. of asthma in Japan. Why it’s not here not to stay well because there’s no money behind it possibly. But if there’s way to generate money, they will bring it here. So that’s where the compounding pharmacy they can take this drug and repurpose it and it’s legal for them because it’s already an FDA approval during the effects and safety profile of I’m not so nice was previously evaluated by a number of studies. And they did the six month toxicology study using dog determined that the dosage of aura Max and offset which no side effects demonstrated is 10 milligram per kg per day. And then it was later determined that that doesn’t have I’m not saying pays for also will be approximately 12 milligram per day for I’m not an ox father more. It was demonstrated that when I’m not saying as demonstrated to rats at doses of 300 milligram per kg per day there are no carcinogenic effect and have no significant effect on reproductive activities on the right. I think they have also human studies because if they’re using it for humans in Japan, that’s means to really track the safety profile for a long time and I think the dosage that we recommend is 40 milligram pill times a day the max I think the main contraindication for administration of amlak Synnex is any noun hypersensitive, so you may have an allergic reaction to I’m not sure looks. Like any other drugs. I mean, we had the potential of having allergy and there are some tastes reported on it from either from locks and also from the ingredients that have been added to it when those you know those ingredients we call it in our care billions are not the active ones. And a case report of 23 year old female who treated for allergic rhinitis with oral sulfa 25 milligrams this guy wrapped a skin rash during the treatment. So what they did that positive test is based on a suspected allergic localized skin basically confirms that she has an allergic reaction to it. You can discontinue this so you can use it in a form of topical possibly, that may override the allergy based on double blind randomized placebo controlled study. For the Q objective subjects. There were no serious significant adverse event that’s a good thing. Two three times daily ordered I’m not an ox, we use almost 40 milligram in the study to patients who experience very vascular inflammatory rash due to reaction to it and proved with local treatments so they have oral allergy to it, you can use it with local treatment, and both have favorable response to the drug. So either you discontinue it or you can change it topical and worse. So give me the recent data that demonstrate the potential of I’m not saying not to bind and inhibit the tank binding kinase which is a kinase protein as eventually they inhibit NF K betta as a treatment to further explore to reduce inflammation, right. And we can use it for many applications that’s inflammatory, in a wide wide range of conditions. That’s why I said you can use it for almost any chronic inflammation out there because it does inhibit the pH one to two to 17. Right. So if you have rheumatoid arthritis, you can use it for you if you have allergy for th two to 17 you can use psoriasis ulcerative colitis Crohn’s disease, all of this can be any kind of dementia or even also the neurodegenerative disease as well. And the beauty of it also is not just inflammatory diseases, but also we talked about and that’s the unique about this truck right, including fatty liver and liver cirrhosis. And this is very important slide. Here is a NPK. See this, this this is the most important molecule or enzyme that we need to know because this protein, this is a kinase protein, and it’s activated by what by fasting when you have to ask them what would happen you would have a lot of asbk Right? Because ATP will turn into APKs. They use those batteries and those used batteries, which is the NPK right that’s been discharged because you remove those two peas, right? Those antibodies they are the one who will activate the NPK and why they activate the NPK. So the NPK will activate the mitochondria to generate energy. Right and activating the mitochondria in cancer can lead to mitophagy and kill that cancer or activation of mitochondria and increasing the biogenesis of mitochondria will help in burning fat. Right turning those white fat into brown fat.
47:58
Okay, and very it’s very important to activate the mitochondria to kill the cancer and a planet xoma Senescence cells so it’s involved in accomplishing, but again, NPK doesn’t have the mTOR and if you inhibit the mTOR you stimulate atrophy because mTOR is mediating the simulation of the apoptosis. And as I said, when you have too big a MBK what you have, you have lipo ISIS and BT oxidation, that’s what we need right? To burn this down, break down the fat and burn it. Okay, slice it and burn it. But in the Andover which is the mitochondria, the mitochondria has been active, ready to go. What else increase the the insulin sensitivity but to me the glucose uptake and the glycolysis decreases the gluconeogenesis and illogical genius. And that’s what we need right? Now, okay
49:02
so what would happen you would have a lot of ATPs generated because of this and because you’re burning the fat you’d be sparing the sugar prop for managing right. And this would help and managing metabolic syndrome metabolic syndrome include obesity, hypertension, I pretension media Botha sclerosis right and hyperlipidemia to cirrhosis and right it’s so there is evidence of its role as an anti inflammatory mediator. And anti histamine we talked about that the drug initially it was the mast still or the Damasteel stabilizing. And we talked about inhibition authentic AB means inhibition of all the inflammatory cytokines and the free pathological free radicals. So it does help to put the inflammation down so it has anti inflammatory effect. Among us a stellar effect the drug is inhibitor of NfkB and the Tbk one, which are kinase protein. We know when you inhibit the Tbk wide and you have it then fkb you inhibit the expression of the inflammatory cytokines inhibit the free radicals generation you took the fire down the macrophage cannot polarize and mature and become m one or m two m 17 which will lead to stimulation of an ad sales to th one or two is to attend 17 Right? So it’s suppressed the whole inflammation across the aisle. And also, when you do that, you suppressing also the interleukin one which also suppressing natural killer cells as well activation so it does help to suppress the innate immune responses and the beauty of this I’m not so nice. They also inhibit
51:16
the fibroblast growth factor.
51:19
Which means I can use this drug to manage all kinds of fibrotic diseases. I can use for corneal scar for glaucoma for the cirrhosis but lung fibrosis for kidney fibrosis for scar or adhesions for aging. As you know aging is fibrotic disease. When we go with growing age, the fibrosis will increase and make us look cranky and aging and our skin wrinkles right now elastic fibers because of too much of the of the collections and the fibrosis and so I think it has an old anti aging effect as well because it doesn’t have the mTOR and stimulate the autophagy inflammasome and senescence cells see how much them Lux and Alex clinical application you can see it you can use it for outrights your inflammation news generation you can use it for dental diseases inflammatory dental disease you can use for cancer, we know why. Cancer two ways. One way is by we know that cancer do have inflammation, but bad inflammation, the inflammation that does not kill the cancer. So they have the inflammation of th 17 mph two they don’t and the th one inflammation being suppressed by the cancer themselves. They secrete chemicals that have the th one is toxic and they are simulating the differentiation of the macrostructure to M 17 and naive T cells T of 17 and th two so what you will do with neutrophils neutrophils cannot kill the cancer you decide which pieces you need each one. So they the cancer secrete chemicals to move the immune system into th 17 and th two and even Tehrik and this will lead to the cancer to flourish and grow because in order to kill it you need to get one and I’m not saying that helps to suppress that perhaps suppress the tears too and it’s something which is a good thing. And then you need to have some other drugs with it. To move the thing into the h1 you can add interferon gamma or thymosin alpha right along with I’m lucky enough so you can move the immune system into th one B cells. But just resetting the inflammation down and suppressing the THD 70 is part of the management of the cancer. So it’s it helps to manage the tumor microenvironment and suppress the inflammation that promote cancer which is th 17 th two and also T reg. What are the things they do in addition to that we know that cancer is some of them are caused by mutation it’s called nonsense. mutation. And those mutations can be reversed by an addition of an MD which leads to expression of those proteins that’s being mutated. And that may help to stop the cancer from or sub those cells from turning into cancer. So it helps in prevention and managing cancer. I’m not saying ox because it helps in metabolic syndrome it lower the limit because it burns a little bit by bit oxidation and mitochondrial lipid I talked about it and also I’m not sure that has anti fibrosis because those aroma it’s not just plates of fat but also fibrosis. And so I’m not saying that says antibiotic effect and make the anti metabolic effects are definitely will help to manage arthrosclerosis And it’s a good thing to manage the root cause of the of the of the hypertension which is arthrosclerosis which is fibrosis and backplates and inflammation but mainly the narrow is caused by fibrosis. So just by its anti fibrotic effect will definitely work for managing arthrosclerosis And that’s the reason I do recommend giving also loads of time because it does have anti fibrotic to manage the underlying cause or root cause of the hypertension. Again, we know it’s managing diabetes and we understand why right guys it goes to stimulate the NPK and because it inhibit the NfkB and decrease the free radicals and decrease the activity of the macrophages but not just macrophages but also the osteo class as the class is basic as Micropress right and eats the bone and by suppressing the inflammatory cytokines and the free radicals. Those two classes will be inhibited and this will lead to Mt use it to manage osteoporosis. Okay, and because of its effect and as I said and turning those nonsense mutation back to normal so we can use it for neurodegenerative diseases, but this is because of its anti inflammatory effect. And it’s managing the metabolic syndrome it’s good for neurodegenerative disease as well, you know, years younger disease is caused by oxidative stress part of it and if you’re able to modulate that and you’re able to express those mutated genes that may be the cause of root cause of this neurodegenerative diseases. Volquez, then you can manage a lot of them like Alzheimer Parkinson disease, and we’re going to talk about in another session of M luxamart. And we know that because it’s managing metabolic syndrome and also it has anti fibrotic effects so it’s good to manage and reverse the artists Croesus, whether it’s from the fibrosis or from the flat prat plagues or whatever, it helps to reverse it right? Because it’s anti fibrotic and also manage their metabolic syndrome. So we can definitely use it for treating cardiovascular. Not just that because of its anti fibrotic effect. You can also use for a lot of cardiac problems, you know, when we had those cardiomyopathy and myopathies caused by fibrosis and it’s because it’s reversing it definitely helped you regain the fraction of the heart so another study conducted 2019 demonstrate I’m looking to Knox when administering cannabis with both intra nasal and oral significantly decreased immune cell response and the cell recruitment. In the lungs, we know why, right? We already spoke about right NfkB these suppressive cytokines are free radicals to decrease inflammation. Why this study in the mouth and also don’t forget, it doesn’t have the locks and not seem like cortisone. Maybe it’s not as as potent as steroids but still it’s dumped those those enzymes, knocks and locks and Cox right. Why this study is the mouse model there was a significant reduction marker of allergic inflammation and similar mechanism to define which is defined as a therapeutic target in the asthma right? And we know as my kids to get 17 Hyper inflammation in addition to master liquidation, so if you can suppress that, definitely you can manage the asthma. So a decrease allergic response by inhibiting the histamine release from mast cells. So it’s a mast cell stabilizer and it was absorbed by inhibiting the cyclic ANP phosphodiesterase. Remember when I talked about that? Just by inhibiting that it helps to stabilize those muscles and the release of muscles another study demonstrate that I’m not sure Knox also all the devils function and patient cell line that have nonsense mutation. This is what we talked about right? And this we’re going to cover it in a very detail when we talk about the next part of flux and oxycodone application. And so I’m not sure how this has been found to not only increase the nonsense containing messenger RNA but also leave a sense of the full length function of protein, which those prevent nonsense mediated messenger RNA decay. So we talked about we said that those nonsense mutated genes, they have this stop codon
59:58
and we talked about those last stop codons are three of them.
1:00:02
You are done. You a G you are away you a and you go away UGA. Those are stop codons. And they because of the mutation they end up in the middle of those DNA for that specific genes. And what happened you end up with messenger RNA with this called kudos. Then this enzyme, which is called the nonsense mediated messenger RNA DK, they will find those messenger RNA with the stop codons they will read them out. There’s a messenger RNA with stuff going on. Let’s go ahead and destroy it. And they destroy this NMD will be inhibited by unlock funnels, which will lead to this messenger night to pass that an override then MD and then what would happen the ribosomes will take this RNA and they start reading it, translating it into amino acids into the protein right? And they will find a stop codon. Yes. But here’s the thing. I’m Locsin ox will cause the ribosome to bind to override it and just insert any amino acid and that’s not good. So you end up with that protein because that stop codon does not have amino acid that’s critical for the function of that protein. So they will put any amino acid there. Just move on. And so it helps to what we call the rethrow. That’s what we call transcription or translation of rethrow. Not just blocking the LMD but also it tools, the ribosomes or close the ribosomes to override the stopping because when they read the stop codon they’re supposed to stop, the protein will stop and you have the corrupted proteins. Well or short pieces of proteins. Well, I’m lucky enough to say to learn about songs when they attach to it, they make it to continue read that and just insert any immunoreceptor
1:01:58
Dr. alasa quick question okay, because I think I’m understanding it better. So yeah, we’ve got stopped codons. These codons prevent the transcription of the of the messenger RNA we we have this this nonsense mediated messenger RNA decay enzyme, I guess that’s what it is that prevents the RNA, the protein from from being translated or produced. And, but then, so So I’m laksa naught and lexan X through one of these the these pathways I think there it’s still connected to the phospho. The esterase sort of lineage pathway decreases the lnd the the non nonsense mediated messenger RNA decay, and then it up regulates what is what is actually putting in something replacing the stop code codon with just an amino acid that is harmless or that will not stop the transcription what is strict will
1:03:18
not stop the translation. So after you okay, you bite me blocked NMD so the MD is not going to destroy those messenger RNA that has a stop codon, right. But then the ribosome will go and read them. And when they read them, they see the stop codon they stop stopping the translation. Okay, that was there’s no equivalent to those tacos and there’s no amino acid that will be equivalent to those concrete doughnuts, you got my point. So they will put they will just put any amino acid there and so override it. override that that stop codon, and what’s also so that is unlocked and locked. So there’s two ways it works on NMD inhibit that, and also works on the ribosomes and override it’s called the repro transcription or translation I’ll read through so the ribosome can read through it and just put any amino acid there because those are positions where that’s been mutated is not important in the function of that protein.
1:04:20
Okay, so Okay, so it’s up regulating the ribosomes ability to do read, read through the transcription
1:04:29
and translation translation of the
1:04:32
Translate translation. Despite the stop codon, it allows it to read it and ignore the stop codon. And this is good because a stop codon is not it’s nothing that is really affecting the protein. It’s a nonsense.
1:04:52
Nonsense. Well, it’s it does not have the amino acid that’s critical for the function of the protein. So you can put it there and move on. Yes. Okay. It makes sense
1:05:04
and it’s put us putting on any amino acid.
1:05:07
Just go ahead, put it there and just move on. Okay. They just get this protein going. Let’s get the manifesting. Oh, wait,
1:05:21
when it’s copying when it’s copying. It’s just putting okay, it’s the stop codon is stop is when it’s copying. It’s just putting any amino acid
1:05:31
what does that I’m not saying like certain way to turn that ribosome and make it to override it. And they will just continue translated the whole thing. Okay. The other word that looks a lot like Bing, that ribosomes to break the law. And that’s kind of Bing, right.
1:05:52
The question was, how does this now let’s consider what how that relates to the spike protein. If we’re going to, you know how the spike protein in the vaccines is causing problems because it has issues with stop codons.
1:06:09
Up to now, I don’t have any studies where the spike protein involved in mutating genes and causing it but we know that the spike routine does induce inflammation. And
1:06:23
we know that mRNA think about this, the mRNA and the pieces of mRNA that are in the vaccine. They have a different stop codon on each end of it. That’s been removed. Right? Is that my understanding?
1:06:39
I don’t have that details about okay,
1:06:41
you can talk about that. Well.
1:06:44
Yeah. I did to prepare a lecture for um Loxone, ox, pharmacokinetics pharmacodynamics and everything related to the pharmacy. The same we did with methylene blue.
1:06:55
That’s that’s no problem that we actually you covered a lot of it. But what I wondered is if we would I just want to make sure that if we start to use this drug, we aren’t going to trigger spike protein related reactions from them or
1:07:07
what we can vote confident we can say that I’m not saying ox because it’s the mast cell stabilizer. As we know the spike protein is a mast cell activation syndrome because they activate the mast cells. And that leads to whole inflammation and the post COVID 19 symptoms. It’s domestic activation syndrome basically and that leads to master to release not just interleukin four but also to Lucan six and mast cells when it’s overactive, it can lead to all this calculation liquidity dimers and the clotting and all that stuff. But the spike protein also activate the playtest directly. So we know
1:07:45
that drug that could be used in that case so yeah,
1:07:47
so I’m not so much because it inhibit the tea at 17. And it because it inhibit the stabilize the mast cells, it can work like Benadryl, still it will be good if they have the acute condition of the post COVID And you have all the symptoms, you need still need that histamine blocker, and then you continue with them. Luxenberg so we cannot really say hey, we can use unlocking arcs in acute condition or post COVID Because some of those patient they have headache, and they have nausea and and they have rash, really still need that histamine blocker. So you had met a drug, but to stabilize the mast cells, which is very important, because a spike protein do activate those mast cells. Then we can use I’m lucky enough but I’m lucky enough to because it screws to press the whole inflammation. So it’s good for Scotland. So I think I will add it as part of the post COVID
1:08:37
We could think about that. Here’s another question for you. Is it not a question but a point? You know, I’ve worked a lot in dermatology and atopic dermatitis is a common condition. And you know, it’s really mysterious when you talk to dermatologists. They don’t really seem to have a clear understanding of it. But you know, they just know how to treat it and they know what it is and how to diagnose it and all that but and how to treat it, but it’s really I just looked it up it’s a th two immune polarization so when you see the word a topic or Tabea it specifically refers to a th two polarization of CD four cells and overproduction of IGE Yes, and what happened? Right that? Well, if we’re talking about that, that the canker sores so the original FDA approval for this drug was for quote unquote, a Tapia, which was not really well defined at that time. And now it’s well defined as a CD for th to immune polarization and causing a hypersensitivity and overproduction of IGE. So that creates the allergic risk of a hyper allergic response. But you’ve really defined what that is here and I never really could clearly they may
1:09:51
add something to it. Okay, there’s not just th two there is also some components of TF 70. Yes, so it’s TF 17. th two and sometimes they end up with a hybrid. And yes, you see the dominant is th two if there’s itching. If there’s no itching, it’s common at the at 17. So that’s there, but they do both. So the asthma is th doing students it’s 17 COVID. Is ts 217 COVID-19. Yeah, condition. So we need to consider also 270 And I’m not getting oxygen habit, regardless of what it is. They inhibit the whole palette, whether it’s intuitive 17 micelle. stabilizer, this molecule I never hear, I mean, we don’t have such Amen. Methylene blue does not do what I’m not an ox does. And now I would like to thank Dr. Joseph James, to bring this molecule on the table for chronic disease. Prevention management, because right now I consider it as a Trump drug. And add to that it will use to be treating 30% of genetic diseases, which we think all those red flags generally, nobody can touch it. We can work on dumping the inflammation and myeloma do all those things. But expressing mutated proteins, this is this is a phenomenon that we can do a lot with patients now with more confidence. The question is that is you doctors here how many Doctor 25 doctors would you be starting prescribing it? And then come back to us and say we find a difference because that’s more important than saying it works. And we have all the studies. But I mean, we still need to use it clinically and we needed for the doctors here to confirm and validate that we did it with with methylene blue with success. And yes, we got all everybody happy with it.
1:11:48
Well, the drug is safe. Dr. alasa you know what you don’t want white How is could you describe for us a typical patient or condition where this might be useful.
1:11:58
I mean, it’s endless. You can use it for neurodegenerative disease, Parkinson, Alzheimer’s, you can use it for multiple sclerosis. You can use it for Crohn’s disease and ulcerative colitis. You can use it for psoriasis and allergy and asthma. It’s blocking a middle it’s suppressed or modulate the immune system across the board. We don’t have any molecule can do that. I mean, there’s only a few drugs out there and they blocked the NFA be most of them they block the downstream of it not the NfkB itself and then as a mass elective stabilizer and then as a nonsense, mediated messenger RNA decay inhibitor. That’s a big, big thing. I can use it for 30% of genetic diseases including cystic fibrosis, good chance syndrome. There’s many other we’re gonna I’m going to list those genetic diseases and cancers. And then next. I will list all this diseases that’s B they have studies for it. I don’t have my mind right now, but I have I’m preparing for it. So the next lecture I would wipe all the list of the diseases that have studies but I’m just saying in general, with the with the mechanism of action, we can use this drug and almost any inflammatory and metabolic disease there is no anti inflammatory drug that is also helping me WTI you can say curcumin does have anti inflammatory in some metabolic and Berberine does the same thing, but not as effective as I’m not.
1:13:42
I do want to speak from experience because I have taken it but that’s what attracted me about it when I started looking at all the different studies related to tank binding kinase and started undernet understanding the mechanisms better is that I took it because of the the the post COVID fatigue like symptoms say brain fog, neuro inflammation headaches, especially allergies, especially during the springtime this year, and sort of battling metabolic syndrome. You know, this, you know, like if I’m not obviously I’m more active than I was, I’m improving, but these things seem to basically I had all of the things that these mechanisms were were modulated by Alexa knock so that that was why I use that the allergies cleared up beautifully. I had times where the brain fog was disappeared. And just knowing that metabolism was was improved. And also I was going to say the you know, with a lot of the biologists that that I’ve had over the past three years, I had a lot of muscle tightness. And you know, like you said there’s an epidemic of fibrosis. Well, we know that you know, when you have mast cell activation syndrome and maybe difficulty clearing the COVID virus. All of these things you’re getting increased expression of TGF beta, which is triggering more fibrosis, you’re having the interleukin, you know for myself on my lymphocyte map I had th 17 th one dominance, very characteristic of neuro inflammation. And and I think I actually did have some fatty liver, so it just seemed like it was handling all of the everything and that’s that’s what really attracted to me to it and kept me digging in deeper to say hey, I think we need to take a look at this. But I did have one thing I do want to mention, you know because you brought up the the the the stop codons, which is which is just takes it to the next level is that now when I come across cases and I get a lot of crazy cases, I have a 49 year old female, otherwise healthy with one that had one natural birth at the age of 40. Who who has a has had issues with with nausea and vomiting, I would say more on average than your average person. And it’s like during her pregnancy and probably before pregnancy. She she’s always had issues with a weak stomach and she finally got some testing done. And a liver ultrasound and MRI says that she has a focal nodular hyperplasia versus hepatic adenomas and here’s the thing here’s what I’m getting at is that as I’m combing through the literature, translating the literature, suddenly I hear that there is a genetic predisposition. Why is she having these hepatic that these focal nodular hyperplasia some of these have pretty big size in her liver. Well, there does it does seem to be a genetic predisposition. And the and they mentioned that it having a stop codon, okay, so suddenly it opens up a whole nother door of Wait a minute, we might be able to do something about this. And that’s the other thing that really energizes me about this molecule is that you know, we might be able to actually do something whereas, you know, you know, that she’s been told by her primary regular doctors that you know, oh, yeah, well just watch it, you know, get an MRI. You know, it may or may not turn into cancer, probably not. We don’t know
1:18:17
Well, well,
1:18:18
we may be able to actually do something. So that’s that’s my point.
1:18:26
Well, great. I mean, I can see that well. Can you hear me? Okay, go ahead. Okay. Two devices, you need to close one of them because you have the down
1:18:52
Are you there? Yeah. Okay, you still close one of the system. One of the invites, your phone or your whatever. You still have that you need to close one of your devices
1:19:11
or turn your volume down.
1:19:20
Jimi Hendrix
1:19:25
Okay, are you there?
1:19:28
Yeah
1:19:32
Okay, so once you fix it then then give you the so I’m not synopsis content produced in either 25 to 50 milligram I think Mike beaver has it been 40 milligram and we know it’s good. For asthma. We talked about it, why there’s a gray nights. And the drug treatment is based on inhibition release of histamine. So that’s where they started semester later. Mast cells stabilize the story. And it also have the inflammatory leukotrienes by the mast cells. And neutrophils, so it does inhibit NF KB NfkB. So that’s mean, all the cytokines, whether it’s th two or TF 17. And so it’s used for allergic rhinitis. I think it’s more than that you can use for any inflammation not just for th two th 17 but also 14 one th 17 And like rheumatoid arthritis and systemic lupus and Oscar Rosa’s analysis of colitis, Crohn’s disease nurse Gill, eco. And human studies study conducted by a more cola, I think is a Japanese because those things are difficult to pronounce. You must say that I’m not saying x x as a bronchodilator or patient with aspirin induced asthma. That’s a new thing. Whether it’s directly bronchodilator like the other day better, you know, they activate the B or it’s working for the inflammation. It seems like I mean, for aspirin used how the aspirin and use vocal construction. Do you know how anybody can remember that mechanism?
1:21:27
Dr. Patel
1:21:32
exactly the prostaglandins Yeah, yeah. Yeah. It seems like um luxon ox maybe will reverse that. And it has similar chemical structure to sodium cromoglycate. So it’s a mess silica Vasia stabiliser, which is known as a bronchodilator used to treat asthmatic patients. So it seems like maybe directly dilate the bronchus, but or it reverse, whatever. prostaglandins that’s been inhibited by that spring. And they started putting patients seven on aspirin and used an eight aspirin induced asthmatic were study inspire measure was performed. at regular time and surveil just showed their fingers efficacy of this drug to reverse that medical use by aspirin but just because it decreases inflammation. And because it’s a mast cell stabilizer that will justify to be managing asthma in general. Regardless whether it’s aspirin induced or pollen induced or environmental induced, I will just give it to all the patients with with asthma.
1:22:40
I did forget to mention
1:22:42
that. Yes, go ahead.
1:22:45
Yeah, I did forget to mention that. The other reason why I was attracted to it is that I was looking for a mast cell stabilizer that didn’t make me tired. Okay, so I do take Benadryl at nighttime. But during the daytime, I wanted something that actually worked that didn’t make me tired and so that was the other thing is there’s no sedation whatsoever. Yeah,
1:23:09
but I don’t think it has direct histamine one receptor block more stabilizing the mast cells. So you still need if you have too much of histamine out there, activating the histamine receptors you still need to have either Benadryl or other drugs. And Old, old antihistamines like hydroxyzine hydroxyzine. It could be an equivalent to Benadryl. And that was so I mean, especially that one I like patient with mast cell activation syndrome, they have insomnia, because this time involved in activation, the reticular formation of the Sonia and it keeps your brain Nice. So you really need to block it. And then you then you can use this to stabilize and as a maintenance, treatment and luxamart so it will be a good match, but still as what Dr. Patel is doing, and she’s recommending all the plastic, no plastic, no aluminium. You know all those chemicals that you can get rid from from your kitchen and the reason because all those petrochemicals that we’ve been exposed, they are massive activators and when you activate your mast cells and you push your immune system to h2 and also th 17 You compromise the immune system to kill the cancerous and you increase the risk of having cancer. So it’s very important to work on your histamine diets any understanding abusive diets or poison or toxins, environmental including fungus infection, those fungus what they do, they also polarize the immune system to th 17 and NPH to inactivate the mast cells and they put you at risk of having cancer. So allergies needs to be managed don’t think oh I have allergies. Okay, so no allergies. People with allergy they have a risk of having cancer because you’re you’re exhausting your immune system to polarize the th two and acetyl th one and so all of this data needs to be initialized and optimized. So us integrative doctors, you know mainstream medicine as much as my work out there. See what mast cell activation they don’t even see it and they don’t even diagnosis It’s a hidden epidemic by the US integrative doctor like Dr. Patel is one of those doctors who are very, that had the insight that health optimization include balancing the immune system and it’s part of the pillar of the of the Chronic Disease Prevention management. So we need to tell the doctors that you need to balance the immune system to minimize the risk of having cancer. And so the four pillars of chronic disease prevention management is four things redox immune, metabolic and then restoring the regenerative status of the patients using stem cells. But just by doing those three, you end up helping to stem just go ahead Dr. Patel,
1:26:14
okay. Okay.
1:26:17
Eco devices so according to 2017 Japan guidelines for adult asthma and lox and oxycontin defined as relieve agent riskier agent may be treating asthma exacerbation due to its histamine integrating priorities and someone prescribing in addition to control agents which are prescribed to control the long term of asthma and is a mediator accurate releaser. So it’s a nice little stabilizer. And the clinical effect of this drug is improving as meta control extend beyond its anti histamine properties because it also helps to 17 and P h2. So it has an anti inflammatory thought that Tbk one has also been noted to postulate insulin receptor and the study and definitely this will lead to lead to insulin resistance and metabolic syndrome and I’m so very important to block the CBK won an order to improve insulin sensitivity and manage metabolic syndrome. And doing that, you can also manage obesity, right because you increase the mitochondrial function, you increase the beta oxidation the label isolates and increasing insulin sensitivity. So the ANP que activation it’s very important which is inhibited by the Tbk one and I’m not inhibit the TV activity one which leads to activation of a NPK and so a NPK. It’s very important kinase enzyme, and I need to have a lecture maybe Dr. juicer or Dr. Patel will come up with a complete lecture on am PK because ANP K activation mediate the mechanism of fasting mediate them mechanism of exercise because exercise increases the physiological free radicals and the physiological free radicals activate them am BK. And
1:28:24
question for Dr. Joseph Shadab. He used them in action. And then he he, what effect did it have on his brain cognition and what happened to his recent memory? Did he have a problem with the recent memory and and it is so then, then, was it better? Because this is a very common problem with the post COVID patient and if it works on the inflammation and everything what you have mentioned in all different mechanisms. Recent memory recall is been a big problem in post COVID patients. So I want to find out from that Joseph that he said he had a brain fog in DD improve with that. And if we did improve, then what kind of problem he had with the recent memory recall and how much it improved? How long did it take, etc.
1:29:27
Well, I mean, all I can say is I’ve been taking it now for about two months, maybe a month and a half. So I did I did have feedback that my speech is a lot more fluent. Okay. So most of the time that I’ve been talking on these zoom calls, especially going back and when I listened to some of them, I could see clearly that I had a lot of word recall problems. And just recently I’ve been told that wow, I sound like I’m talking a lot more fluently. I’m talking faster. I’m not stopping or slipping over my words. But that’s been the big problem for me over the past few years. But what I can’t say is that other things are not contributing to it, because I am trying out some antifungal products. I do have evidence of mold toxicity in one of the the array 12 lab tests which is associated with sirix labs, and I do have like symptoms that would be connected to or that would point towards mold toxicity and possible fungal overgrowth. So,
1:30:45
memory lapses,
1:30:48
memory lapses, whether you mean specifically
1:30:51
like like, you know, what did you do today? And then it just takes you a while before you answer, or you just say that, no, I don’t I don’t recall. And then you later on, you remember, oh, yeah, this was the question, and this was my answer, but you were not able to say when you were asked, or when you were you were put on the task at the time. Yeah, that’s,
1:31:17
that’s pretty common. Like things I would normally remember somebody asked that just can’t seem to remember and these are not necessarily difficult. Things. It’s like, like, even people and people’s names and I’m like, gosh, what is his name? And, you know, that was that happens a lot. And as far as my mental state, you know, when it’s when the brain fog is bad, okay. It feels like everything is in slow motion. Like and I cannot seem to focus or direct myself to get things done efficiently, and everything seems very, like difficult like simple things seem difficult. And learning new things. incorporating new things seems difficult. If somebody were to say, oh, you know, you just got to get that that app on your phone, and then you can get your answer there. For me, it’s like, Oh, my goodness, no, I’ve gotta get the app. I’ve got to sign up. They probably want to answer some questions, and I’m probably going to mess it up. And I don’t want to do it because it sounds exhausting. Well, that’s ridiculous. Because the reality is that that’s that shouldn’t be complicated. That shouldn’t be difficult, even if, maybe if there’s a few things, technical things that you have to figure out, but for me, it just seemed exhausting, absolutely exhausting. A couple of times, when I did use Alexa Knox, especially the first time I was having one of those really bad brain fog days that it just seemed like nothing would work. And I took the Alexa Max and within it took about two to three hours. And all of a sudden I felt normal. Okay, so you know, I take these things as little evidences to take along with me. And look for these times when, you know when it appears, a small miracle happens. Right.
1:33:34
So because one more question that all other problems you had, like you said, you had a mold problem, mycotoxin problem, etc. But was that the COVID was the last one that broke the camel’s back.
1:33:52
That’s what I think. Because when I when I got my first lymphocyte map, in 2022, I believe may have I think that’s when I got it either at the end of 2021, or the beginning of 2022. I had evidence and I could show it to you. My total lymphocyte count was about 1000. And my normal is, at least according to the Sirex lab was 1200 to about 3600 My CD four count, I’d have to go back and look was close to about 300 Wow. Three 300 That’s that’s far aside. That’s aids Right? Or that’s right.
1:34:38
When you say that, it’s less than 500 They’re in trouble.
1:34:43
Exactly. That’s why, you know, when I tried to explain to people like, look, I’m sick. It doesn’t really register because I might look okay, you know, because I’ve gotten all the different peptides and different things that I’m doing but my it’s like, that’s what broke the straws back. It’s like all of a sudden, it’s like yeah, what happens to AIDS patients they start getting opportunistic infections. Yeah. Right. And so suddenly, my symptoms turned into the symptoms that seemed to be involved with with mold toxicity, because they seem identical to me. A lot of the mold toxicity symptoms and post COVID fatigue. They they overlap. In fact, I’m beginning to wonder if post COVID fatigue has more to do with with fungal overgrowth and mold toxicity for people for people who were there. For which their symptoms are going on longer than a year.
1:35:46
Know that you have to pay more attention to your mitochondrial function. And you have there is a special mitochondrial, tacked down. And if you start taking that along with the mitochondrial peptide and try to do that, and at the same time, you you are taking because you this is what is going to help you with the with the mitochondria and like Dr. Eliza pointed out that if you do the NPK and you put that up, that would be good. So I think that what what you are saying that you took the amlak snaps and then you started feeling better, you know, how many blockage you removed and how many blocks are the inflammatory cycle you put in NF kappa B and B 17. Aisle six out for whatever you blocked and and that would have a fact. I still think that nobody has talked about endothelial dysfunction and vasculitis and that is the part of this process. And it and I think that we should talk about these The to address the endothelial dysfunction, because if we talk about that, because yes, cladding is secondary fibrin monomer is secondary. But if our school it is and that vasculitis we have to address and an intra Asian car accident I think the vitamin C and other molecules do play a great role. We have seen patients with the anaphylactic shock after the COVID vaccine which works similarly, food sensitivity develops chemical sensitive develops, and we cannot explain everything on the basis of the mast cell activation. And we could see that that would be the histamine and if you take care of the histamine didn’t work on her the prednisone superhigh those worked for a while, but then she she developed osteoporosis from that before she came to me. So I think we are all going on the right track. But there are some more points that some that we need to address. And And always remember, martial activation is a piece of pie and it is not the whole thing.
1:38:32
Hey, if you right now real quick, Dr. Joseph. Have you have you heard of you see so much Vedic. So Ma? v e, di C?
1:38:50
No.
1:38:52
Okay, this is with electromagnetic frequencies. They’ve got devices that they’ve created. To put in your office and put in your home to regulate and down regulate electromagnetic frequencies. And they have a lot of what do you call patient? That it’s absolutely help. I think your electromagnetic frequency sensitive and they’re one of the best. And there’s another one that I’ll get to you after I figure out you know where they’re at and so forth. I really think we need this in our office and we need this in our home. Okay, it’ll like break up and if you get shung guide, if you were shung guide, put it next to your heart and you were shung guide and you put it in your pockets and you put it in your office it’ll regulate the electromagnetic frequencies, because they’re off the charts. They’re really off the charts. They’re like radiation, and it’s very intense. And I think you’re sensitive, just like I am and Patel had mentioned that to you last time. So I think this is a big part of what’s going on with you. And if we can, I want to get they have anywhere from I think 800 to $5,000 for high level gold, you know, devices to low level devices. And I’ll talk to Dr. Clearfield and we’re opening up a clinic right now in South Reno, and we’re going to be working on this type of stuff. I think this is way huger than what all of us think, and I’m gonna do my best to you know, get this type of information to all of us. Yeah, Joel
1:40:47
it’s funny that you brought it up because there are EMF measurement meters available. So before you go to the extensive stuff to buy it, you first find out what what kind of EMF you are exposed to, and where which part of your houses or which part of your offices are high, because we are all surrounded by it. And then more chemicals and mycotoxins and the the stuff interactions you have. They are the biologic factors which make you more susceptible to develop the electromagnetic sensitivity, but by lowering your load, you can do it but but the avoidance is the main main treatment and how to know that you are exposed to it or not. And and so by this method, that meter is about 434 $100 And if I have in my office, and we can we can give you the phone number address, and you can get that mirror and measure it. I think it’s very important to measure it before you do the corrective procedures and you can even find the patient digitaler is sensitive to or not, bye bye, bye. If you have an EMF free field, and then you bring the doctor phone or something to patient and patients start having symptoms. You know that patient is the EMF sensitive and then you can be sensitized it. You can buy the equipment, all the all the different even even they have a night night canopy to sleep at night and you surround your your bed man with the canopy. There are things that you can put on your body that things you can wear an undershirt and so on. It can be a big full lecture, but I’m just giving you the few tips and you can take on from there I lectured at a big conference EMF conference in in Santa Cruz just few years ago,
1:42:55
and I absolutely agree doc. You can you can have mats underneath your feet. You can mats underneath your iPad underneath your computer. You can have mats underneath your bed, and you can ground yourself the whole time. It’s absolutely amazing once you start doing it,
1:43:14
yes, great. Yeah, no, that’s a that’s a great point. And I’m always looking for the next level of things. And it’s true that the you know, I became me in that sense if I could, it was like a perfect equation. If I got into say a car sat in the backseat of a movie called the car so that the popular electric cars I can’t think of it. Tesla Yeah, I mean, I could just feel the symptoms. It’s like HERE IT COMES THE it was almost like bloating, brain fog. And it’s like, oh, this this heaviness and stiffness and my upper neck and shoulders. Like wow, I’m like, Man, how am I going to deal with this? This new world we’re in and I can’t tell you though, or my ears will be ringing but I can’t tell you I have you know, as I’ve sort of started well, here’s here’s my main point. The cool thing is just like with an M lexan. That’s, you know, the purpose is not to say M lexan axes is everything. The purpose is to say, You know what, we’re getting more and more targeted and refined with how we can modulate these these dis regulations. And so that collectively, you know, first of all, you know, perhaps we won’t have to be on 3075 different supplements every day, as you know, but we can we can we can have take fewer things and but we know exactly what we’re targeting and and we’re accomplishing more with like, one treatment modality. And of course we use other treatment modalities. And like, yeah, address EMF and and that we should go about the EMF with a similar mindset that we’re not necessarily shooting in the dark or like you said, Dr. Patel, you know, maybe we measure it to to see, but we come at it with a targeted approach. We think about it the same way we think about how we’re thinking about am Lexa Knox and yeah, you know, and what I have been doing has been, has been helping dramatically, but but Joel yeah, thanks for bringing that up. I’m always happy to hear, you know, a new company or new approaches. And and yeah, I believe that that is very powerful in as as another piece of the puzzle.
1:45:57
I’ll do my best to put the information on that. If you don’t mind putting your information in the chat. I’ll send you that and I’ll send you also another German company that has very powerful mobile and home processes to really remediate EMFs. I’ve been following it quite extensive. So if you don’t mind, you can you know, DME or whatever.
1:46:25
Yeah, absolutely. We’ve got the mastermind going here. So in our just take it all in and learn from each other and the beautiful
1:46:34
thing. So let’s just finish up those little slides. What’s left is all pictures so just showing here I am reluctant I’m not going to knock it doesn’t have it that an FK B are those kinase and eventually it will inhibit the mTOR and this is very important for killing inflammasome very important this is this is proptosis. And so when you block them Tor you induce apoptosis, and you use autophagy of the senescence cells cancer cells and and for inflammasomes. We know exactly the NPK which is very important to manage because folic syndrome and healthy cells, possibly I’m not saying ox as well. As KB KPV. They activate mTOR which helped to stimulate the stem cells and the healthy cells but in pathological sales, they activate them mTOR and they lead to autophagy. So that’s a very important point, especially when we learned from KPV we find out kbv does it stimulate them tore into healthy cells, so that’s good for treating retinal pigmentosum.
1:47:54
Please go back to the previous slide. Okay, yeah. So if you look at these mTOR and look this proliferation, migration invasion, and this is for cancer, yes, yes. This isn’t all the vectors so you inhibit them to they’re just good to kill them. Then it works in the same principle. works in the tumor microenvironment is very important. So when cancer cell this is, this is on a healthy salary talking or in the under cancer, so
1:48:26
we’re talking about mainly cancer cells mTOR will be inactive and ANP K is very good in activating and active and activating them for and this is a mistake here. I think it’s anti apoptosis. And so if you inhibit mTOR, you induce apoptosis. And you can have all of this in the cancer. So and also in inflammasome, and senescence cells, so activation of mTOR it’s part of getting rid from the inflammasome cancer cells and the senescence cells. But I would argue that mTOR can be activated in the healthy cells. So there is a little bit different response, healthy cells versus cancer. So the mTOR and why because healthy cells stem cells you need to activate them for to stimulate the stem cells,
1:49:11
because Madison mTOR is very important.
1:49:15
Right? So So it’s different here as far as county sales versus cancer versus cancer cells. inflammasome is senescence cells. And their response to those drugs are a little bit could be opposite, like curcumin, stimulate the mTOR in the stem cells, but inhibit the mTOR in the cancer cells. Got my point? Yeah. Okay, so it’s really important to know that and BK it’s a very important molecule. I hope Dr. Patel Dr. juicer will come up with a lecture on amtk because it shows all the mechanism of action of exercise fasting, all those things it stimulate they and became a NPK. When stimulated, they stimulate the NFR to another knock to is expressing the antioxidants right. Although antioxidant enzymes is expressed by activation of NFR which needs detoxification is an antioxidant, but a NPK does stimulate and FR and we know they have BK what’s been activated you end up with the increasing the mitochondrial biogenesis and health increase the procure to ISIS, which is very important to even killed. The EMI Lloyd’s possibly and every cycle that for that’s a good thing about a NPK activation may helps in breaking down those bad proteins which is prions. And also it helps increase the energy balance we know that motivation the mitochondria and increase the burning of the fat right and increase insulin sensitivity here. And this definitely slowly crafters of the aging, right and optimize the health just by activating asbk And most of the polyphenols works by activation of the NPK physiological free radical that’s induced by actually sighs activate the NPK which will definitely activate the NFR again, I am PK it’s very important see exercise here a low am aamp which is empty batteries. Activate the NPK here and it leads to the burning of the fat right? We should have different the acid synthesis burning it up anabolism so it’s increase the fatty acid catabolism which is fatty acid which is beta oxidation, right and induce autophagy by inhibiting mTOR directly activate autophagy and we know it’s inhibit them for right and this will lead to autophagy and let’s go on to more about the inhibition of those protein kinases. The Tbk one and I take a E which leads to inhibition of NF gay and they did the randomized blind control study and they find it working for managing diabetes and we know why because of the stimulation of the NPK and definitely when you stimulate the NPK you break down the fat so increase the lipid lysis and the beta oxidation so you manage the fatty liver here. Treatment of patient produced reduction of hemoglobin a weight see that’s how you know it’s working trials to show that there was a reduction and fractals are mine. And this is very good because I think it’s a promising drug for weight loss and for managing metabolic syndrome and decrease the inflammation. I think it’s it’s a good track. I think Mike beamer is doing 40 milligram versus 53 times a day 12 weeks a use of it will definitely help to increase the insulin sensitivity and decrease inflammatory markers giving us potential effect and longer range of effect. I’m not saying that should continue to be explored with regard to its immunomodulator effect. We talked about it and I think we are using it for asthma, right and for diabetes and obesity and fatty liver and I think all of this we have studied and applications behind it. And we know that you know one of the hidden problems that we have is mast cell activation syndrome, and liver fibrosis fibrotic diseases. I think this will help to manage those two, pandemic hidden disease which is muscle activation. syndrome and fibrotic diseases. Were other drugs they cannot really do much. And I think it’s medically metabolic syndrome, right. All the three things diabetes, malaise, dyslipidemia, hypertension, obesity, and we need to have a serious investigation by you start using it on yourself and your patients and coming up with your results and feedback as we see with Dr. Joseph.
1:54:07
Do we think it will work on a biliary cirrhosis?
1:54:10
Yes, it does. Oh, it will work with biliary cirrhosis. In fact, possibly one of the abilities that reverses is also the nonsense mutated genes. We’re going to talk about it but yes, it definitely helps to fibrosis in general, because it does inhibit the th 17 but also inhibit the fibrotic growth factors. So based on this data, anti inflammatory agents we can say and like I’m lucky enough to have anti inflammatory anti histamine helps in metabolic syndrome helps allergies and suppress the chronic inflammation in diabetes and obesity and absent metabolic syndrome right. And we need to investigate how we investigate for you to start using this drug I think the safety profile is very high. Dr. Yusuf is using it and I think it will help your patients and we need to learn from you again NPK very important enzyme. I mean then when you go to a for me hear about mTOR mTOR. mTOR is a DA downstream and it can involve yes then if you inhibit it, it definitely stimulates autophagy but if you stimulate it, it will help to stimulate the stem cells. But this is our bloodstream of HBK doesn’t have an mTOR all the way there and that’s very important to know. And the stimulate the autophagy and those inflammed inflamed and we know that it helps in burning the fat right lipo lysis fatty acid oxidation, stimulate them toward or directly stimulate autophagy and increase insulin sensitivity and the ability cause uptake. And this is very important for managing metabolic syndrome. Again, it’s a similar to NfkB and NfkB does the same thing. It didn’t have the NfkB the sorry, the KVP KPV, which has been assessing ledingham and peptides and I’m like Xanax both of them. They didn’t have the NF K b and both of them are very good adding to the NFA beak and the sorry the T V KKKP V which is you know something around peptides. That’s how anti fungal effect and anti microbial effect they make holes and those those fungus and destroy them. So adding luxon ox with with the BV is a good idea. So you can get rid from the fungal infection you know does that bacteria because I don’t see I’m not seeing as antibiotic effect or prebiotic effect versus TVB that so I will encourage adding to the M Loxone ox kbv so you can get rid from the from the fungal side and also it helps you to protect from the electromagnetic waves because those electromagnetic waves have their own pathological level and constant and that’s what we exposed and when we go to the conference with all this wiring or inside the car of Tesla, they they open up calcium channel and that calcium channel will lead to opening up the mitochondria will release those free radicals pathologically. And you end up with pathological free radicals because of the electromagnetic waves. And you can buffer those and detox your body from those pathological free radicals by giving the unlocks and I’m not sure if it’s good to prevent or manage oxidative stress from that electromagnetic way but if you have all the other tools to measure it and prevent yourself from exposing it to those constant EMF which is an oxidative stress agent or free radical generator machines. But again, there is you can create system that heals from the EMF. If they are producing Christological level of free radicals. That’s why we use EMF dead and all that stuff, too as a healing but if it’s constant exposure that will lead to pathological theoretical non physiological and that will inhibit the ANP okay. So in physiological those one that healing ones the EMF do stimulate the NPK because it generate physiological level of free radicals versus the pathological exposure that you have and and the Tesla car industry, there are hotels or resorts in in Vegas where there being all the building wired and you feel sick in it. And the reason because of this constant exposure to the EMF which leads to constant pathological free radicals that will inhibit the am BK. So again, I tend to use both the locks and locks and kpb which has been isolating among peptide in managing all this disease here autoimmune cardiovascular metabolic organ joint disease, pulmonary diseases, you know, disease, cancer, right? And mechanism of action of metformin is activating the MLK mechanism of action of Berberine polyphenols, all of them they activate the NPK and we see how they implicate works in breaking down the fat and autophagy and have to definitely kill the cancer. And it does stimulate the expression of antioxidant enzyme it also expressed the NLS, so it does help improving endothelial function. I think Dr. Patel talked about it and just by activating MBK, you can you can improve them to to function. That’s why how the polyphenols work but for my words, I’m Luxton ox works the KPV work by NPK. So I think we need to have a lecture of a NPK and talking about in detail. Again, we mentioned that I’m lucky enough because it didn’t have the NFA and NF kappa B. Then it’s good for treating osteoporosis because you are inhibiting osteo class we don’t ask the class as a form of macrophages if the bone and if you can block that. And you block all those cytokines. Especially if you’re new constricts and all that, then you can decrease osteoclast activities and you can treat patients with osteoporosis. So it’s a good thing right? To add that in your equations. Again, I think this is this shows you again the amtk and the metabolic syndrome and how they are generated from activation of this i ke B which is the NfkB at eventually and how this inflammation and all of this leads to activation of bacteria infections and all that stuff leads to activation of this NfkB and we activate the NfkB definitely you lead through insulin resistance and you lead to metabolic syndrome and you manage it by blocking the anak NfkB by blocking the ache I take AB which is the which will inherit the NfkB again, you see that the T the t v k and NfkB NF kappa B, all those kinases, definitely it turns or it it caused a tumor to progress. Why because it’s stimulate mTOR right. And mTOR will lead to proliferation, differentiation and invasion as you see it here. And it will lead to stimulation or release of those cytokines, especially the interleukin six, same interleukin four and it goes the polarization of the immune system away from th one and it will change the tumor microenvironment in favor of the tumor growth. And in addition to that, we know the stimulation of to the Tbk one or the NfkB will lead to stimulation of the fibrosis right. And all of this can lead to the tumor progression and invasion and amlactin arc so anything that didn’t happen in fk B will be helped because it will help to suppress all those bad inflammation which is that he had 17 inflammation associated here. And it will help to possibly move the immune system to th one and also it will block the formation of the fibrosis which squeezes the blood vessels which prevent the infiltration of the lymphocytes from killing of the cancer. So that’s the reason I think, an addition of CBT one it’s part of management of cancer. So I can definitely give a Black Sox or other patient with cancer, but I will add interferon gamma i will add I will add the immune immunotherapy which is anti Pdl, one anti CTLA four, I will add a vast in all of this. I will add the metabolic therapy redox therapy and photodynamic therapy in managing cancer. I will add last time as well to really cancer from all different direction including low dose chemotherapy. Again, this is so so easy when when the macrophage gets activated by the LFO by the lack of polysaccharide from the bacteria, how this will activate those protein kinases which is the Tbk one and that leads to increase the MFK betta here and that leads to the expression of those inflammatory cytokines as you see it here, which includes interleukin six and leads to polarization of the immune system to get 17 and inflammatory response. And to block that you can just unlock some oxygen habits this those kinases, right.
2:04:01
Again, and again. This is a shame thing just shows you how inflammation start and how this inflammation activate the DBK one maybe throw those like receptors when they are receiving those bacteria infection and how this will lead to activation of the interleukin six and polarization immune system into th 17 And all this inflammation that you see here. Again, for managing chronic disease, we recommend all of your doctors to be trained to load those drugs into platelets nanoparticle. Because if you do that, then you can get those medication to the pathology and we have the training for that I myself I trained many including Dr. Patel, Dr. reduces their hearing and they witnessed the effect of playtest nanoparticle. It’s a very good tool. It does enhance and then you can add everything inside your box but let’s say you want to do a joint inflamed joint osteoarthritis, you can add your oxytocin you can add we were going to have an Luxton oxygen, Luxton ox, sterile, you can add your loads of time and you load inside plate and under particle you can add procaine and oxytocin and all those drugs that you think will help in your joints. And it stays longer in the joints when when you put it in the form of BMP or you can put it it can give the patient’s IV they say having cancer or you’re having it, you want to deliver it into the brain. You can do it into nasal you get the speed and then a particle delivering those drugs into the mythology because nanoparticle size, in addition of tagging those peptides and molecules and preventing it from being degraded, when they are circulating in the blood, it stays longer in the body and then because the pathology is leaky versus the healthy so you can get those drugs. Going into that pathology and other things, the platelet membrane receptors, they have this natural receptors that target the inflammasome the senescence cells and the cancer cells. So you have a specific targeting not not just passive targeting but also an active targeting. Again, you can also
2:06:17
go backward and back. You mentioned about not this one, next one. This one, no, no forward, you’re going backward. Forward, okay. Yeah, here yeah. Yes, yeah. This injured blood vessels. So what what are you targeting here?
2:06:44
Well, we have injured but this is part of injury. Even part of clot if you have atheroma or the Roma, for example, and you want to target dose with the drugs that breaks down the fibrosis or helps to decrease the inflammation so you can know your nanoparticles and it gets there and you start they start releasing into those injured
2:07:13
nanoparticles with
2:07:15
what the drugs that we know that injectable that would help in arthrosclerosis Those are 10 is one okay when we start having the injectable part of it that would be a good one.
2:07:30
I see. I’d say keep Towson for sure. Now keep in mind, I mean, the growth factors from the platelets, the platelet nanoparticles also have have the growth factors. I believe that would help with the the endothelial growth factor.
2:07:51
The platelets growth factors in fact, it’s it’s going to be another side of the sword, but it just delivering those drugs that’s of interest. Anything that helps to thin the blood. No, no curcumin, there is injectable curcumin. You can load it inside your plate and nanoparticle may help to get them there. And then the blood what are the things that thins the blood and breaks down the fibrosis?
2:08:13
What about methylene blue and in light?
2:08:17
Yeah, I mean methylene blue does work for everything. I will definitely add it and there and anything that helps to Iran’s
2:08:28
N acetylcysteine.
2:08:31
Not only good, yes, NAC will be a wonderful drug.
2:08:34
That’s gonna help because if we give a higher dose that will help with clotting, as well. And it will increase
2:08:42
oxytocin methylene blue, if you want to. There’s any streptokinase that is not as powerful that you can add or anything that breaks down the platelets. That’s not her cominius But if you find anything that helps maybe low doses trip to kinase, maybe 10% of the regular dose, if you want to add it there that you know that will not go
2:09:04
here baby peptides should help right.
2:09:07
What do you want to do?
2:09:09
KPV
2:09:11
KB v will be good, yes, but not the well. Now the band. The other one is BBC, BBC 157. Do we have injectable for that? Yeah. Because that will help. For patient who has blockage in the cardiac, you know, angina and they have arthrosclerosis and narrowing and you want to develop collaterals because it’s stimulate the end what is the vascular endothelial growth factors? Right. So yes, it does. It does
2:09:46
work up regulates the gene of the interest cells.
2:09:52
So it helps the building the collaterals and that’s exactly what you need for patient with angina and you have this blockage or arthrosclerosis which is fibrosis along with some five green and some black of fats. So we want to break the fibrosis those are 10 will be good. And you break it, you loosen up right along with kbv will be good too, because KB V and M Lux, anoxic we develop injectable for this, that’s our anti fibrotic drugs will help to open up those blood vessels right? And loosen them up. And then then you also want to build collaterals and I think that K BBC 157 If you have the injectable one peptides would help you build up the collaterals Yeah, so I think those those are things that you need to you need to be critical thinking and just don’t lose the dough is the big deal just as 10 20% from beginning to see how the responses and then you can go from there all the time. Use loaders, because you think there are plugins that are critical will enhance the delivery get with the pathology, so you still get the effect. And if you use the full dose, you may have any side effects that you don’t want. So even just 10% upgrade is still only getting better results. If you want to do stem cells and we do that mean Dr. Joseph James we train people to do fast sell our transfer. And you’re in the process of fat transfer when we do the micro
2:11:15
cells in the Nano.
2:11:18
You can you can you can one the
2:11:20
process of the train systems are better than stem cells. You can do
2:11:23
exosomes if those stem cells are stable enough to stand the sonication and the nanofiltration. That’s fine. But isn’t that stem cells that when we do the fat transfer and we do the micronization of the fat and we get into the face those micronized it’s right there. Why right you can ultracentrifuge it like putting it in 5000 Round 10 minutes and you can get all the stem cells being squeezed in the bottom and then you can take that and you can suffocate it and add drugs if you want to use fat derived stem cells and nano used as a nanoparticle to deliver drugs. So during the time, well, we don’t really when we do the training, we don’t want to say we’re doing fast stem cells extraction. We said we don’t fat transfer for cosmetic procedure, but down down down to the I mean, while we’re doing that you can take those micronized It’s fat and because they are being broken those stroma and released those stem cells and you can just centrifuge it and you can get the stem cells and you can use it for treatment if you want. Again, this is also from the stem cells that can be used the same thing like we do a plate with the plate is much cheaper than stem cells so it’s not that wise to use the stem cells if you have the platelets for delivered drug it’s much cheaper than than stem cell but if you want to be creative, in other words want to do extra
2:12:47
have the membrane to write. Stem cells do
2:12:50
yeah so we can use stem cells.
2:12:52
But what about exosomes?
2:12:55
Well, let’s his arms will be also good. The question is that are they strong enough to stand the sanitation and then a filtration? I don’t know. So but it’s
2:13:06
better than stem cells are not. They are more right so basically
2:13:09
don’t why we don’t just skip the skip the sonication or do syndication but never do a little of syndication not by just to make drugs on occasion not to break down the exosomes and just go with the with the none of the exclusion process and the exclusion itself would push those drugs into those, those exosome by diffusion. So with them those exosomes with sonication do a little authentication, just to make the drug not to break them, and then do more of nanofiltration
2:13:42
I don’t know if this will help but Dr Clearfield adds BPC 157 to his semaglutide with I believe it’s vitamin B six, and that gets rid of the nausea. So I don’t know if you guys can work with that. But that’s what he’s done and it looks like we’re having dramatic results. We got one army veteran. We lost 36 pounds in six weeks. no nausea, no nothing. And he’s doing great and I think it might minimize the thyroid cancer, possibility of thyroid cancer. I don’t know if that’ll help.
2:14:15
Let me tell you about that. I will not touch I will not get BBC narrative through the cancers and the reason because it does stimulate the basket at the growth factors. And those will push the immune system to three rate and it has the, the which we need to that’s why we get a Baston which is the opposite of what BBC will do. So very important that we know what we’re doing with cancer and not putting peptides that may aggravate the cancer. I believe EDC is one of them by analogy and vomiting all that stuff. It’s good. That’s an anti inflammatory. Yeah, that’s that’s okay. You got my point. Yeah,
2:14:59
got definitely.
2:15:00
Right. Okay. So we’re good today. Again, I will invite you to attend the Chronic Disease Prevention management Weekly Webinar seven 3:30pm. Every Monday, please attended and we’re going to repeat this in our session. This is the first by the way. He got the first lecture, Dr. William. Oh, I’m Luxor Knox. So he’s the first even before even our Mondays so we my group they don’t have I mean, it will be new for them. But if you want to join me in add to this it will we want to repeat it again and my session and I think we’re done. Any questions? Was long lecture right Dustin
2:15:47
Go ahead. James, and I have been working on my leg I have a personal blockage of one hour I did have a partial blockage of a blood vessel and leg probably I felt due to scar tissue from an injury. And we treated it just using orals and you know, because we had time I was not particularly symptomatic. I had monophasic I had mono phasic flow in it. It was very narrowed. We use serrapeptase and endo calyx Pro which improves glyco calyx and red light therapy and also am Lexa Knox for any inflammation. I I had several things that sell that I could not get back into his office. I went to his office and I had been treated and treated for about three months. And I I now have multi phasic flow he wouldn’t commit to whether it was biphasic or, or tri phasic but it sounded tri phasic to me and look pray phasic to me, and so that blood vessel is now open and and he wouldn’t even dream of treating it was thinking of standing up before. So you know there’s an oral treatment with red light, which worked very well.
2:17:26
Oh, yeah. The light therapy does increase the nitric oxide and helps improve the need to function and a light therapy is amazingly and if you add the methylene blue and you add all the tools that you have, I mean come on. Dr. Joseph James. I will consider him right now. And along with Dr. Patel. They are the frontier of medicine and things that they have. It’s it’s the top
2:17:47
so I have a patient with PVD and here’s a here’s a stent, but but he probably has the atherosclerosis everywhere, although may not have symptoms, so I’m treating him currently, but I thought more can be done. So you think the methylene blue can help with the red light therapy,
2:18:13
along with Alex and Alex along with the KP VkV Yeah, I love all of this just because there’s an anti fibrotic and artists there. osis is not just removing the clot and removing the aroma. It’s fibrosis. So those GaBP and I’m Lux and ox are anti the best anti fibrotic. I mean, we saw that it’s good for liver fibrosis. And, and so if you add this in along with the light, which improve endothelial function tests because it will prove the increase in nitric
2:18:48
oxide, here’s a hypertension. So I have to be very careful with the methylene blue Yeah,
2:18:53
just lose use low doses and you’re okay and then with light therapy, and when you do those are 10 as well. Yeah,
2:19:01
no treatment would help him any more than the
2:19:05
think it’s better to pump articulator to get those medications and especially if we get the injectable closer cannon but you can still use oreillers or 10 five milligrams see how they were, I mean, whatever he does with hypertension, just switch into those are 10 Whatever we what he has a potential. He’s on TV. Yeah, you said one of them is another pro right? That one you can switch them with less than a pro because it’s that’s what we do. When we have any side effect. We’ll listen which action to ARB and the cardiologist will not be upset from that. And yeah,
2:19:43
I was gonna I was gonna add something my mother had mentioned, Dr. Dow. Well, first of all, I mean, her vascular surgeon, of course, he wanted to do a stent, but you know, I thought otherwise. And he thought otherwise to once we were able to show that she she didn’t get a pulse with with these modalities, but I just wanted to add another thing that we could talk to into the future just something to not a mystery er. But she was talking about the endo calyx and glyco calyx. It’s actually the active ingredient is actually for Croydon, which is a very powerful condensed Japanese I believe Japanese seaweed that happens to modulate or upregulate certain sirtuin six Okay, so what I’m starting to see another pattern and things to look into as you know, are we are we turning on certain sirtuin genes in this kind of is is close to aamp K as well because I think once we get these sorted out,
2:21:00
NAD stands on the sirtuin genes. If you give any IV NAD it does
2:21:12
not serve two and six. It’s served to in one I think three and seven don’t quote me but not sir two and six or two and six is is is very difficult to to activate and that one is involved with DNA DNA regeneration and fixing the DNA. So in cases of microcirculation that has been damaged to a certain point there needs to be a regenerative you know that’s where we get into the the other leg of the pillars of chronic disease. You know, we’re Buffett we got the redox we’ve got immune we’ve got metabolic but then we have to up up regulate regeneration and fixing of the DNA. And that’s my understanding. I’m just saying this this is just something to add we can go into in depth into the future, you know, with multiple articles to in pathways to demonstrate what we’re talking about, but we’re talking about microcirculation and there’s there’s actually been they did a study on COVID patients in the ICU showing that the there is a direct correlation between the the condition of their overall condition of their micro vasculature which can be measured and their mortality. So you can imagine that the micro micro vasculature which is really where the endo calyx lie live, so endo calyx are the sort of little little hairs that the you know, that are involved with the microcirculation and function that that we lose those with micro vascular disease and especially for Pat, if we’re having like little micro clots. That’s another arena to to look into. And obviously in my mother we also injected PRP with with oxytocin into the scar tissue as well. That was another thing that we did so anyway, so that that’s what that’s all about. So yeah, for Croydon sirtuin six and I’m glad you brought that up Dr. Patel, because yeah, and ad turns on, especially certain one, but I think I think at some point we should have a learning about the sirtuin genes in general since they are the master regulators are ultimately the master regulators of metabolism, inflammation and regeneration longevity genes that if they if they’re turned off well then the aging process speeds up.
2:24:05
Can I Can I ask a question or make a comment? Well, when you have exotic birds like a parrot that lives for 33 years or a cat contato that leaves for 20. Fatty Liver Disease can kill them. And it’s painful to lose them and when we went through this, our vet recommended lactulose and I’m thinking that Alexson X might be good with it or better than it and then when you have a large dog that you love very much they can get hip dysplasia and Dr. Sylvan talks about hydrocortisone and maybe M lexan. X might be worthwhile in that regard. Other than also the Aussie My daughter has note swims a lot. So I wanted to mention that and the other comment going back is for liver spots and the skin or age spots. This might be a panacea to some extent
2:25:23
are you talking about and Lexa Knox being a potential treatment for age
2:25:27
spots on the skin? Yeah, age spots, liver spots and you know, certain creams, whatever. But you know, blood centers can mess up your skin too.
2:25:40
Yeah, I mean, that’s that. That’s a good question. Now. I can say because I do have a cosmetic background that you know, when the certain spots on the skin, they may require, you know, some sort of a laser sort of shattering of the pigment to get rid of it. But then of course preventing it from happening. I could see how the the pathways involved with and Lexa Knox may be playing a part. It sounds like redox is really important. I know that vitamin C helps as well because there’s there it does. Redox is a part of melanocyte overexpression if there’s too much oxidative stress. I do know that one for sure. But yeah, some of these spots they they need to be broken up like shattered in order to get rid of it. But yeah, I think the but but yeah, in concert with some of these other strategies make sense.
2:26:54
You know, losing a pet is not a pleasant thing to go through. So that’s why I mentioned the birds and you know, our daughter had her first thought he did only live till seven years old and he was he was able to keep them alive through periodic you know, couple blood transfusions a week. So that was not fun.
2:27:20
Yeah, and that’s why I love that, that lecture that the there was a veterinarian that gave a lecture a couple months ago which was fantastic. And basically utilizing all of the things that they’re they’re using at wellness centers, hyperbaric chamber, even pulse electromagnetic frequency. And yeah, I mean, I have no problem with giving my mother’s dogs. They’ll some of the peptides, you know, we if his hips hurt, we’ve given them BPC 157 I think TB 500 and actually had them had them lay down on the pulse electromagnetic frequency. And literally, you could it could literally walk and run around better and jump around and go up and down stairs like dramatically better. So yeah, we can use these treatments. On dogs. They’ve got the same you know, basically the same molecular chemistry as we do as far as I know
2:28:18
where the dog sleep at night is very telling you know, they’re they they sleep in the bedroom around the bed often. People
2:28:35
Yes, they do. I’ve got Do you want to? Are you ready for questions? Dr. Alaska.
2:28:42
I’m open if there’s any questions. Yeah, so if not, then
2:28:46
yeah, no, we got we got a few questions. Right. Now, let me see. Number one is see what kind of Contra indications that you know,
2:29:02
allergy allergic reaction to it like any other drugs. So that’s the only thing that’s been reported. It’s very safe. It’s been used since 1993. Up to now. There’s nothing we reported. That’s that’s that’s alarming.
2:29:20
So andalo RX is a common common one sodium Mimic, or vice versa. Says so ama MLR. Next is a cromolyn sodium or vice versa.
2:29:38
It came from this man this my stabilizer como glycans, you know, cromolyn whether it’s a modified it’s not completely same, but it came from that drug.
2:29:52
Okay, and Dr. Joseph, what dosing did you use on yourself? Yeah,
2:29:58
great. Question. 40 milligrams three times the day is needed. So 40 milligrams is the dose that that I used, that I still use.
2:30:09
So you’re making as needed unless you didn’t take it every day?
2:30:13
Well, I took it every day. Yeah. But you could use it as needed. That was the original script, but I felt better if I took it three times a day, especially because allergies are just ridiculous over.
2:30:26
I don’t know if it’s okay for you. But that’s the juice if you have psoriasis as a background of multisystemic disease and this is if you take it every day. You’re justified to take it every day. But for people with anti aging or healthy people, you know, they can take it as needed and possibly just take it per week, twice a week or something just to give that break of cycle but people generally in general when they have underlying inflammatory diseases. I’m okay with any questions? Yeah,
2:31:02
yeah. Dr. Stoker as the process Stan Glen dins Landon’s. I’m not sure what that is about.
2:31:13
What’s happening with all the land ends,
2:31:16
not with all the land as he was just being funny, I think with the echo.
2:31:20
Okay. Gotcha. Gotcha. And then Dr. beamer said very interesting drug. Thanks, Dr. alasa. Dr. Joseph for the info and Dr. Clearfield for hosting. I have mo next I needed to give me a text and I will get any fo you all need three to 13010752 Okay, so Okay, got it. Try now is there any Benadryl league to dimension?
2:31:48
Our offense? What does that does that?
2:31:51
Is there is there a Benadryl lead to dementia? There was some
2:31:57
study about Benadryl use for a long period of time. That’s why you know, you don’t use it all the time. But people with allergies, they’re using it all the time. And there’s studies about because it has anti muscarinic effect that may lead to dementia. But at the same time, there’s studies in Harvard Medical School and others that is good for preventing cancers. So it’s a sort of a to edge. I would recommend using it per need and not religiously to break the cycle of any harm that’s coming from it.
2:32:33
Okay, double question from aura. Did you try given your gut a rest and support in Dr. Terry walls, protocol, mitochondrial support, I would think would help. As you put a link in the
2:32:50
past, of course, anything that helps to stimulate de NPK which is all those everything actually goes most of the time, activate de MBK polyphenols works so MBK I will recommend adding whatever helps to stimulate the mitochondria. That’s good. All right. No.
2:33:14
Certified herbalist give me no parsley mix to drink it work. Okay, that was Dr. Morrow fits. Shell Stein C four s t.org. She put that in the link. More fits. Send it to me Joel Okay, got that. Blah, Blah. Blah, blah. Some of Vedic Okay, that was me. Shell Stein. EMF. Joel mute everyone. Okay, so I’m trying to find this solution. That’s me. Let’s discuss this later. Shangai more fits fascinating. Where are these doctors based? The drug doesn’t exist on good acts. Okay, Clearfield Marino
2:34:01
can answer that? Yes. Right. It’s not a good reaction because it’s compounded. Next
2:34:09
Okay. Long term steroids destroy everything. Skin eyes bones can help reverse.
2:34:18
Yes, if you activate the MBK by M luxon. Ox or polyphenols. I think it will help.
2:34:27
Okay, thanks, Dr. alasa. Excellent. As always, dogs are people to Dr. Stoker Pavlov. Yeah, I agree. decided to put my service dog down who used to be our mascot at doc Clearfield after 16 years. That was so hard even as a Marine shell aside Pavlov had it wrong it’s the dog that conditions the master. Amen to that. Dr. Stoker, he needed it every day. So it looks like we’re good.
2:34:55
Okay. Do you know this is the longest lecture two hours and 34 minutes? Amen. Absolutely. Yeah. Well, great. Well, hopefully what happened center in Hawaii Right. Dr. John? He’s still working on that.
2:35:12
Mute is I want to permute is I’m just saying I’m just going to interject. Go ahead. Dr. Burgess. It’s all Yes.
2:35:20
We’re working on anything goes right now. We’re just looking on ideas. West Coast, East Coast, any subject, any place. So Florida is possible. And if you guys if we want to get just a little bit rich, we might want to go to the Bahamas that wherever we go. We need to be careful.
2:35:39
They have 1000 square foot. They are people with top doctors possibly they want me to run that center. And if I do then I will bring my people to be there like Dr. Patel Dr. juice if you both of us, we can create the model of what we’re doing. You know, including Dr. William I mean, he is a very he’s the Western metabolic part of the Chronic Disease Prevention measurement. And Dr. Patel in the cancer thing and he’s in her immune modulation. With Dr. Joseph with I think he covered all the topics. We can create the model. Then we can replicate that mold everywhere.
2:36:20
So are you are you talking about the Bahamas?
2:36:23
No, I’m talking about Florida there was a group. Okay, they have 8000 square feet. And I’m going to be there in Florida next week attending their conference speaking there, actually. And then I’m going to be in the beach, the condo there. So if you want to come and have fun on Sunday and swim with us, because I have lots of meeting with those business people, possibly the the guy who’s putting a big center near to Orlando, the 280 acre Medical Center and condos and resorts and all the good stuff. I’m going to meet him again. And he’s interested in our model to create the you know, the protocols for his medical center that he’s building up. There’s a lot of potential everything’s still in the air. But if we land on something, then we can replicate it and we can work together on it but without having a model on the ground. With CPM and what Dr. William is doing and what you’re doing and having all this joint force in one place. And that replicated it’s gonna be difficult because it just, we are everywhere.
2:37:37
Well, let’s communicate.
2:37:40
That let’s let’s reach out to physician assistant artment. He’s in Florida, he could probably help with this whole process and I’d love to get there myself just saying
2:37:51
well if you want to meet me there in Florida having fun anyone but maybe Stefan will come because not far away. And me and Dr. Joseph James. We’re gonna have four days in the doctor class. You know the road. And in the I mean in high, you know, the high condos. I mean, those are one of the top ones right? Where’s this? From February from June 14 15 1617 1819. So 14 to 19. But Sunday it’s in the high than the Hollywood beach next to Dr. Class. Oh, Hollywood. Yeah. Okay. Okay, this Sunday, if you don’t have anything, just come in. Have fun. Yeah, sure. Anybody anybody wants to meet us there? Welcome. Yeah, that’s that good.
2:38:46
We’re gonna stop recording.
2:38:49
Okay, well take care.
2:38:51
Good night, everybody and see you next week.
2:38:54
Good night. Thank you. Great.
2:38:56
Thank you Doctor all Lhasa Thank you. Bye. You bet. Bye everybody. Bye bye
2:39:10
Okay, I think I’m supposed to close this out Dr. Break
2:39:16
when we started the recording, I somehow shot to my computer. So I turned the recording off to think that we would be a little safer and getting recording. We’ll see what happens okay.
2:39:28
Yeah, no doubt. No doubt. I was dude. The traffic was off that it took me 45 minutes to drive nine miles in Reno, Nevada.
2:39:40
We gotta think about where we, you know, we got so much incredible stuff going on now and the situation in our country is becoming more and more unbelievably radicalized. I know. I know, to a stupid point. And anyway, we we have so many we have Dr. McCulloch who is an angel who they tried to turn into a devil. So have about 10 other people who were angels, they’re trying to turn into the devil. So, so so many subjects we could talk about right now. And
2:40:21
we’re, we’re in the middle of Okay. Real long story. Short, Dr. Fung. He’s one of the top line specialists. I believe in America, definitely in Nevada. He wasn’t paying his rent he got into I believe he’s bought us burgers, and he messed up didn’t pay his rent. He got kicked out why. So his friend, Darren, who was a contractor in Iraq and Afghanistan and South America. He came in to help him and that that all fell apart.
2:41:00
I remember that so that didn’t go through. No, it
2:41:03
didn’t. So I brought in Jennifer Baker. She is a powerhouse here in Reno, Nevada, and she’s on the legislative team. And she’s she knows everybody. She knows billionaires, blah, blah, blah. And we’re also best selling co authors in the same book. She took over she went off on Darren. He hasn’t paid a couple of our doctors, okay in advance practicing registered nurse who’s also homeopathic. So Jennifer Baker now took over, went and talked to Dr. Fong. He got kicked out for a whole bunch of reasons. And so now we’ve taken over his business and all his equipment is still there. So for the past two weeks, it’s been like, chaos, total chaos, and I’m going back and forth before between Dr. Clearfield and Jennifer Baker and Jennifer boscovich Jennifer boscovich. She’s the advanced practicing registered nurse, and she’s a homeopath. And then we have Dr. Wolf. Dr. Wolf is a homeopath and an IRA Vedic doctor, who was trained in Greece, India. And Israel. And he also knows Jewish medicine. That dude is a freakin same. Who is this guy? Dr. Aaron Wolf.
2:42:28
Have you know he’s in your area?
2:42:31
Yeah, he’s in our area. He’s actually he’s homeopath. And our Vedic. He started out as a doctor, and he was at UC Davis when his dad caught cancer, and they overdosed him with chemo and radiation and killed him the next day, straight up. So he straight up just left went to India and now he’s our Vedic and he’s homeopath. And he’s absolutely intense. Okay.
2:42:59
I also, I also got a doctorate in homeopathy through the United Nations. I also studied era Vedic in India. So a lot of the things we talk about I’ve been there, I’ve done that. I don’t, I don’t really want to do it. But I want to be an advocate for those therapies. Because they’re a Vedic was half of all the treatment at the largest hospital in India. It’s like acupuncture being used in China. So we have all all these unbelievably wonderful methods. I get it, I get it. The question is, how do we introduce this this without without having doctors be headed? So every time you use the word homeopathy, I shudder I deal with a lot of stuff and alternative medicine, herbal medicine, acupuncture, nutrition, but if you start going down the homeopathic line, they will kill you. It’s like the biggest war the AMA has ever had. 150 years of fighting homeopathy, and they have not stopped. And the supplies we use for a pain integrated pain. prolotherapy neural therapy, the supplies we use have been blocked from the United States. It’s like you think you can run your car on gas and then they take away the gas. So homeopathy, I’m very, very interested in I do I practiced I have a doctorate in it, but I am scared shitless to tell the truth. So anyway, this Aaron Wolf, you talk about you and I both think he’s got to be the greatest thing since sliced bread. But he, you know, he will be but he will be attacked, and you and I will be killed in the crossfire. So if we’re going to be doing something to advocate and benefit him it’s going to have to be very clever, very clandestine. And you know, then I would love to support it, but coming straight out and supporting somebody of that quality. It’s going to be just like Dr. McCulloch like Dr. Malone, Judy Mike of vets, Carnage, they’ll try it. Sherry Tenpenny they’re going to try to rip them alive, man. And you know it. But anyway, that’s why I want to get together and talk to you about this stuff. How can we have these meetings and the organized organization expand, you know, to our benefit. What else I was going to say something else about I forgot what it was. But anyway, I just want to move really cautiously with this because you’re telling me of the finest of the fine and I appreciate it like as a gift from God. Because usually people like that who run to the Himalayas, India, learn era Vedic, learn homeopathy. They’re motivated by something different. And once you once you discover air of Vedic and you discover homeopathy, you can never be the same person again. Because our whole thing that I think we can win this game with Joel is the theme and the theme is ethics. And the ethics are three parts and that’s why we have three pillars for our logo, safety, efficacy, cost, era Vedic homeopathic is the highest level. That’s the most beautiful, safe I mean, talking about safety, that’s what we do. And what I do and I’m gonna write a book on because it’s basically already written I’m gonna steal it from my mentor, but he he developed told me a puncture. So here’s a perfect opportunity to combine acupuncture with homeopathy, which gives you an added potency to the treatment, but it’s still but still, it’s still say it works. And it’s the lowest price medicine in the world. homeo puncture is the cheapest method of medical treatment on the planet Earth, and it’s safe. Okay? Now, the only thing is, is it efficacious does it work? The key to keeping our organization together and this is what I’m planning here with the medical school is a search. So we have to have those in our group who want to play research. And that can be surveys. It can be all kinds of things. It could be double blind PLACEBO, and our plan is to do that out of the United States. We have row a tan Honduras, which is an island 20 by 40 Mile Island where we can do the research. With other we basically wrote an island is a dive center, the second largest reef in the world. So we have 12, maybe 20 dive shops there, and my partner down there, probably can go into any of the dive shops. And then work with them with surveys and research. Okay, so our aos i am is going to be protected by research. The research at the medical school will probably be directed toward me a little bit, and then I’m going to direct it toward the Caribbean a little bit. So we don’t have to deal with a bunch of federal bullshit. And the FBI coming in like they do it did with our group and the AOA said make them disappear. We don’t like Bill fear. Clear, Ill Joe or that Burgess mother fogger. This is what they do. And I’ve been there they just they just took a fortune away from me in our community and they let 1000s of people die. They are now dead. In my little hometown, because they saved seized our facilities. Gave me the demand, keep your mouth shut and get out of the way. They kept moving the dope through the area. There are bad boys bad boys. So I can’t take any more of that shit. That’s why I want to focus on research because we’re telling the truth and out of the United States, or they will suppress us too much. And we’ll never be able to do anything. But if we’re out of the United States, we’re playing our own game. That’s bliss. That’s my plan. And where do you say you want to have a meeting Florida where?
2:50:08
Okay, right now let me back up the CBD and let me write this down. Okay, we’re pin look, Nevada is the only state in America that has a medical board for homeopathic medicine. Right?
2:50:23
I understand that and you leave Nevada you come out and they cut your balls off
2:50:27
these debts. I know I know. So. Jennifer Baker. I’m a best selling co author with her in the book mastering our mountains. She knows the legislators she knows the congressmen she knows the senator she knows the governor. She knows freakin everybody. She’s running the whole clinic. Okay, Dr. Wolfe doctors any nurse practitioner Jennifer boscovich, who was a 16 year emergency. She was an emergency nurse. She was a death nurse. I forget that forensic nurse. She was like really intense. She’s a badass. This is the team we’re developing right now. And so with the homeopathic medicine, we are connected to the homeopathic medical board. The last one in America. And Dr. Clearfield is getting ready to be certified. And someone else I think, and then Arizona. They don’t have a medical board. They have a medical group or whatever. So we’re like the last group to be fighting this whole medical thing and I absolutely agree with you. We need to go abroad, dude. The only reason I’m still here in America is because of Dr. Clearfield you Dr. Baker Yeah, no, no yeah. Yes and no. Okay. You know, so doctor or nurse practitioner boscovich and doctors any you