Tuesday Integrative Health Webinar
Tue, May 30, 2023 4:59PM • 1:58:14
SUMMARY KEYWORDS
vagus, ehlers danlos, patient, connective tissue, frequencies, treat, eds, joints, pain, genes, hypermobility, fsm, vagus nerve, danlos, years, people, problem, inflammation, hyper, week
SPEAKERS
Bill Clearfield
Bill Clearfield 00:04
Alright somebody got admitted so I don’t know if that Oh, wow. Okay, yeah. All right. It was I was on the wrong one signed signed back in so still make me I got the Yeah. Hi. All right sorry. Everybody I was on I had the wrong was on the wrong Zoom meeting so my my my fault so hopefully don’t lose too many. So tonight start tonight Joe I’m gonna make you co host okay.
00:56
Yeah it’s still it’s still telling me
Bill Clearfield 01:00
you have another meeting and progress tickets. I think it’s led me and looks like it’s late and you should have let in. Yes. Okay,
01:08
got it. Okay, I’m gonna make you co host okay, because tonight our speaker
Bill Clearfield 01:20
is not going to be available at six o’clock. So we have a video that we’re going to look at. It’s her webinar. On ailleurs Ehlers Danlos Syndrome. So again, first of all, I’m sorry, I was on the wrong Zoom meeting so I hope we didn’t lose too many. The So Carol McMahon is she was on with us last year. I’ve spoken in her group a few times. Her group is the frequency specific microcurrent it’s a it’s a type of device that’s a it’s kind of like a 10s unit electrical current and they’ve matched different frequencies to different organs, different disease entities, and they have had some fairly good success with it. It’s pretty interesting. She I think I got the story right that she had gotten married to an osteopath an old osteopath. And this was in the 70s and they want a practice in somewhere in Kansas I think or Nebraska. And in the room was a was a this in the place they bought was a device that was an electrical stimulator. And it came with frequent different types of frequencies. And they started playing around with it. They’ve been using it ever since. And she’s pretty good. She’s got a pretty big following. She’s got about five or 6000 practitioners. She teaches all over the world. This technique, I’ve been threatened to learn about myself, but I always have something else to do. And she was on last year with us. She’s going to be on at six o’clock to answer questions, and she can explain it a little bit better. It’s a little bit after five. So what I have now is I have one of her webinars and this is when ILAs Danlos Syndrome.
03:26
And I hope
Bill Clearfield 03:29
you all have a at least a smattering of an understanding of what that is. If not, I think she’s going to explain it. And I’m going to we’re going to just
03:41
we’re going to
Bill Clearfield 03:48
let Dr. Nereo in first and we’re going to get rid of that. And we’re going to let’s see what else do we need to do we need
03:59
to start our recording I think the recording started already. So let’s find the video
04:15
and here we go.
04:17
Okay, this is a dare Ehlers Danlos webinar, and this is what we’re going to be talking about
04:23
for the next 65 minutes.
04:30
Why don’t we treating so we’re going to go into more than you ever wanted to know about airless danlos and hyper mobility spectrum disorders. I didn’t even know there was a hypermobility spectrum until Tuesday. Details about the connective tissue configurations and the genetics. What are the tissues from an FSM perspective? How do we treat these similar syndromes? What are the risks of treating or not treating, and what can we expect and how do you do this? That’s the plan. So there’s 13 subtypes of Ehlers Danlos. Then there’s the HSD, generalized peripheral, localized and then historical. So that’s older people that were gymnasts or really hyper mobile when younger but they aren’t now. So there’s actually a way of scoring this it’s called the Beighton score, and apparently there was something different Carter and Wilkinson. So this is sort of a patient version of this. Can you put your hands flat on the floor with your knees straight? You score yourself. That’s, that’s one. Bend your elbows backwards, bend your knee backwards, bend your thumb back, can you bend your little finger at 90 degrees
05:52
so this one, number five, and it’s
05:57
I don’t know they say little finger but I I’m used to using the index finger and it goes past 90 degrees in the patients I’ve seen so 100 and 100 degrees 105. It’s pretty extraordinary. That spandex finger so maybe the little fingers is more difficult to bound. So when it gets to 90 degrees or 110 It’s really
06:25
a thing. So
06:27
this is from the website, Ehlers danlos.com EDS is group of genetic connective tissues disorders resulting from college and defects are the defects and proteins that interact with college and so it is a multi system disorder and as you’ll see, there’s going to be a lot of different symptom presentations from just bendy and hyper mobile to lethal. No two patients are the same because the connective tissues that are affected in this system are different depending on the patient’s so it was named less than 100 years ago.
07:08
There are still large portions of patient reports that are unknown.
07:20
Sort of teen subtypes I had no idea. Many people with EDS look healthy making the disorder largely invisible. And this is from their website.
07:32
It’s on. There is no cure. But
07:39
as you’ll see, at the end of this hour, there’s a possibility that FSM changes this so signs and symptoms, body pain, dislocations subluxations, which is heart
07:53
partly dislocated pipe vulnerability,
07:55
arthritis, osteoporosis, and you’ll see that that arrives because of defects in the collagen. That whole bone together if you saw John sharpies lecture last year
08:08
came demonstrated that bone is calcium filled fascia.
08:18
Bone is fascia. So when you get difficulties with the collagen that forms the bone, you get osteoporosis, skin tearing stretchy skin, really soft skin because it’s missing the collagen at the bottom mitral valve prolapse. Easy bruising, fatigue, Kyary syndrome sprains GI issues a trophic scarring that scarring without a lot of ridge of connective tissue and muscle spasms poor healed look at this list pots, organizers rupture. patient dies of an aneurysm. Did he just have
09:04
arrows danlos
09:06
prolapse irritable bowel dysautonomia, flat feet, Swan that deformity gastritis joint pain muscle pain, surgical complications because the thing won’t heal. So collagen is everywhere and the genetic changes that result in a diagnosis of Ehlers Danlos by a geneticist is
09:33
different for everybody. And where it
09:36
shows up in which kind of connective tissue is in which tissue. We don’t actually know because nobody studies this stuff because there’s no drug that’s going to cure it. So this is a slide you can just go back to but it lists migraines
09:55
or Thursday orthostatic intolerance pots,
09:59
mitral valve prolapse, it’s a lifelong condition that you were born with, but I’ve had emails from two different patients that say that there are infections
10:13
by Bartonella mycoplasma that cause
10:22
Ehlers Danlos in later years and you’re you’re born with it, but it doesn’t always show up in the milder forms. So I wonder if the infection basically affects the vagus which causes inflammation which makes the pain short. So there’s that classes so this is the 13 types. Classical Ehlers Danlos skin hyper extensibility. So if if your patient can do this with their skin, or this with the skin on the back of their hand, or That guy’s sort of famous on the internet, generalized joint hypermobility, so this is the Beighton nine Ford score again, it’s autosomal dominant. So if mom and dad had it, the kids gonna have it. And it’s these are all have to be confirmed with molecular testing. So three centimeters in the neck, elbow or knees, one and a half. centimeters for dorsum of the hand digital forum, one centimeter for the palm of the hand, but I couldn’t find a picture of that. So main symptoms of hypermobile EDS, tummy problems. So that’s the digestion we’ll get to that at the end of this dislocation stretchy skin. Easy bruising, why would you bruise easily? That’s because the basement membrane in the arteries is made of connective tissue. So if that connective tissue in the arteries is defective, the arteries going to lead more easily. bendy joints pain and you’ll see that I think that comes from the spinal cord, fatigue. feeling dizzy, why would that happen? Because the ligaments and connective tissue and
12:14
tendons at the upper neck are
12:21
touch with connective tissue to the vertebra.
12:24
highest percentage of
12:27
proprioceptive is in the body or the suboccipital muscles. So those muscles and ligaments are at C one
12:34
- lacs. They see one is going to move and those muscles are more stretchy and they have still have the same amount of appropriate centers. So this condition doesn’t affect the appropriate centers. It just affects the connective tissue.
12:56
So that’s why you feel dizzy. So they may look like they have a vestibular injury, but it’s a cervicogenic vestibular entry because of the stretching acid sequence.
13:07
So this little girl is our she’s our target
13:13
gastrointest gastrointestinal issues. We’ll see why Potts, chronic pain fatigue, easy bruising, hypermobile joints, TMJ, osteoarthritis poor healing, prone to injuries, the patient’s history includes these kinds of things. I want you to start looking for Ehlers Danlos because it now
13:36
is something we can treat.
13:40
And the problem with EDS, as you’ll see in a few slides is that they don’t find it because nobody looks for palpitations, lightheadedness, dizziness, fainting, elbow joints, right joint pain, and you’ll see where that comes from hypermobility mitral valve prolapse ortho stasis and sinus tachycardia. Okay, and we’ll see about that. Classically the s, this one’s different. It’s autosomal recessive, non dominant, and it’s 10x B gene. And this is this is the easy bruising. So this kind
14:23
of it EDS
14:27
is associated more associated with bruising, and then there’s the Beighton score for hypermobility.
14:34
Google,
14:37
Ehlers Danlos and you’ll find all sorts of cool stuff. That’s where all this came from. cardiac valves, hilarity EDS. Apparently the connective tissue is specialized in the heart valves. So it’s severe progressive cardiac valve problems aortic and mitral especially skin involvement. hypermobility in the joints, Autistics. autosomal recessive, but you notice it’s a different gene. That’s the one a two gene this which is different than the one that’s in classical so these are genetic testings that are done by the geneticist that you send your patients to valueless vascular EDS, it can be severe can be life threatening. There’ll be a family history. And the gene associated with this is the CLL three a one which I don’t know anything about. Arterial rupture at a young age. Colon perforation, uterine rupture during third trimester in the absence of previous C section or severe Peri partum, perineum, tears. So this patient went through her life, at least until menstruation or teenage years, got pregnant and nobody knew that she had
16:09
Ehlers Danlos until her uterus ruptured
16:15
during the third trimester of pregnancy, excuse me. I mean, this is really fascinating. And this is autosomal dominant so somebody in the family should know that the person had carotid cavernous sinus fistula is those are up in the
16:33
neck autosomal dominant,
16:37
confirmed by molecular testing this patient is going to need to see a geneticist It is rare, very rare, potentially fatal. And the facial characteristics Look at that. You wouldn’t guess that this kid had something wrong with him. Small chin. Big guys.
16:59
long eyelashes. What’s up with that narrow palate, midline flattening, deep set or almond shaped eyes? I mean, who would know? Right? Interesting many sleep with their eyelids partly open. They have easy bruising. So very superficial veins and easy bruising.
17:26
Vascular Ehlers Danlos so vessel ruptured aneurysms, so there’s see the aneurysm
17:32
right there. So you test for it with well you send them to they get that vascular study
17:44
and with contrast and radiology and you send them to a geneticist. At the end of this webinar, you’re going to know more than most people about Ehlers Danlos hypermobile EDS is fairly straightforward, mostly generalized joint hypermobility and then feature a feature B features C and two or more of these have to be present in some combination. All these prerequisites must be met absence of skin fragility, including auto rheumatoid, autoimmune rheumatoid arthritis conditions.
18:28
That may also include
18:29
joint hypermobility by means of loss of muscle tone and connective tissue, last laxity. So, these prerequisites have to be met. And there has to be these other things that don’t explain it.
18:44
Okay. So this one
18:48
doesn’t have a specific gene associated. This one is obvious at birth, bilateral hip dislocation, severe joint hypermobility skin hyper extensity extensibility. It’s rare. It’s autosomal dominant. So these genes are starting to look familiar now. Right? The cog all gene which is collagen 181, or one a two, and this is diagnosed, obviously at birth, and by a geneticist.
19:22
dermis, der Mattos Berra, I can’t even pronounce this ones. Are axis
19:29
EDS or D EDS.
19:31
Nine major criteria 11 minor criteria to anyway it’s autosomal recessive. And I didn’t include pictures I think because the pictures were so creepy I couldn’t put them on. But it is a different mutation. And these people end up so the cranial facial facial features. Yeah, this one ends up but the geneticists to this one becomes obvious by the time the child is I don’t know. Looks like two or three congenital lacks muscles congenital or early onset kyphosis scoliosis, which suggests it’s going to end up in a chiropractor’s office but look at this
20:17
because there’s pictures
20:20
generalized joint hypermobility with dislocations subluxations peripherally, so shoulders, hips and knees in particular is autosomal recessive. So neither mom or dad has it. But the uncle they never met and if Dad was adopted, brother that he didn’t know about or an uncle he never met.
20:43
And these are different genes
20:47
again, hallo D, which I don’t know what it is. And for these, these people ended up with the geneticists. So if you’re a chiropractor and you see somebody that looks like this, don’t even go there. Don’t start adjusting the spine. Can you imagine what you do to an Ehlers? danlos patient when you do a high velocity thrust, same thing with the Osteopath and even the PTS. It’s like seriously, you start mobilizing these joints and they’re going to dislocate. That’s not a good thing and that’s fine. So there we go. So cornea syndrome, this one thin cornea.
21:28
The other pictures on the internet were
21:33
kind of too creepy to put on here. So I didn’t. It’s autosomal recessive, which means it’s harder to find mom doesn’t have it. Dad doesn’t have it. But his great uncle had it. So it’s a recessive gene. Mom’s grandmother had it and it’s recessive. And by the time you get down to
21:58
the kid, nobody knows that it’s in the family. So this is what they look like. And it is
22:07
confirmed probably by an ophthalmologist when the kid has initial trouble. And then you send
22:13
them to a geneticist, short stature,
22:18
progressive and childhood. This one is obvious at birth, and by the time the kid has two or three, muscle hypotonia bowing and blends they’re going to end up in a physical therapists don’t think they’re good. If they look like this. They’re going to end up in the chiropractor’s office. But pediatricians in the group will see this autosomal recessive these are the genes involved and these people end up in the geneticists office, and then you have to figure out how to manage them to make their lives more comfortable. That’s what I’m saying. We can’t fix these are genetic, but the potential exists that the FSM protocols can help these patients, musculoskeletal musculo, muscular contractural, eds, the pictures on these it’s really rare, so it was hard to find photographs of the patient’s multiple contractures abduction flexion contractures or clubfoot. cranial facial features that are evident at birth are in early infancy. And the pictures I saw on the internet were not particularly characteristic. You wouldn’t look at that kid and say, Wow, she looks weird. I think she has this rare kind
23:41
of Ehlers Danlos. But
23:46
so characteristic cutaneous features skin hyper extensibility, easy bruising, skin fragility with a trophic scars and I’ll show you what you saw what those were before. Increased polymeric wrinkling. So there’s the skin on the Palms is more wrinkly because the connective tissue that should be strong in the palm of your hand and not have a lot of wrinkles when you do this. There’s doesn’t do that. It’s sort of folds in on itself and wrinkles. It’s recessive, which means these patients aren’t going to know that it runs in the family myopathic This one is really rare, congenital muscle hypotonia muscle atrophy that improves with age so they’re pretty messed up as children. But as the muscles develop and they have more growth hormone, the muscles develop but they’re still going to have proximal joint contractures. So see this one two out of the 11 people known to have this kind. Okay, so hypermobility of distal joints, so
25:02
her knees, hips and elbows, contract but her fingers and toes and ankles are hyper mobile
25:15
autosomal dominant, or autosomal recessive, which is kind of be confusing for the average geneticist. And it has that collagen 112 A one so their collagen is everywhere in your body. So there are a lot of genes that are responsible for this one for the dentists in the group. Severe and intractable periodontitis at early ages, childhood or adolescence and the key for the periodontitis is the gingiva the gums don’t attach to the teeth. So seeing where the periodontal ligament is how the tooth is constructed. See that. Okay, so there, that connective tissue that makes the tooth and holds the tooth
26:13
to your gums. isn’t made right.
26:17
There will be a family history of a first degree relative who needs to clinical tried to and it is autosomal dominant so it’s easy to find. So if mom had a dad had it, Uncle Ralph has it. You they know that when the kid’s teeth starts coming in and the gums bleed. They know that it’s genetic. And I mean, that’s the good news. And the the bad news is there’s no way to treat it. So hypermobility spectrum disorders are similar in URL or downloads but there’s no gene identified. Because there’s so many genes, similar patterns, pathologies of the airless danlos group, but there’s no clear genetic markers, and the pathologies aren’t as severe, but they involve similar symptoms. So you’re still going to have digestive issues, you’re still going to be diagnosed with fibromyalgia. You’re still going to have joint pain either because of the discs and the spinal cord, or because you actually have your joints bend the wrong way, or bend too much generalized H S D. hypermobile ability spectrum disorders, many hypermobile joints peripheral just the hands and feet, localized hypermobile and a single joint or group of joints and for our purposes, we’re going to treat it as the same as rose danlos. Same pathologies and mechanisms. It’s just they’re just not as serious but I suspect they’re more common. These are more likely to be missed. Because it’s not such an obvious life threatening mass historical
28:13
age HSD
28:15
older people who are hypermobile one younger but aren’t now. So these are the people that come in to see you. Now older has a very wide range can be any place between 40 and 74. Right? They weren’t. They come in for neck pain and low back pain.
28:36
They were gymnasts in their teenage times.
28:41
They were ballerinas up until the time they were 25 or 30. And a ballerina is considered old.
28:48
And they’re very hyper mobile, which is helpful
28:55
in their profession and their applications, their hobbies. But when they’re adults, older they have facettes syndrome. They have disc injuries, extremity joint involvement caused by the Joint trauma when you’re a ballerina, or 25 year old gymnasts so we’ll treat the joints facettes dis, the vagus auto immune issues, inflammatory issues and guts issues. These are going to remain after their joints are more degenerated. They’re not as hyper mobile. So the HSD disorders have all of these
29:42
symptoms that go with them and the patient walks in with pots.
29:47
You’d never think I never think to check them for hypermobility. I can think of three patients right now that I missed. Never thought of it. Chronic pain, fibromyalgia fatigue is the version of a patient a friend right now then
30:02
moved to Australia. And women find out she had Ehlers Danlos until about what two years ago. Arthritis Look at that. Now
30:16
the joint hypermobility is the presence of too much movement at any given joy.
30:22
When it is part of a chronic condition and cause cause varying levels of physical disability,
30:33
it cannot the hypermobility can occur with no other symptoms and be advantageous if you’re gymnasts. So nobody cares. If you’re a ballerina at the age of seven and you can do the splits. Nobody cares. You’re a cheerleader in high school, and you can do the splits.
30:49
That’s not a problem.
30:53
Joint hypermobility as a part of a condition that causes pain and fatigue like fibromyalgia
30:59
part of a chronic condition.
31:01
It can be accompanied by GI dysfunction. We’ll see how that works out. Headaches DHA C one C two or hyper mobile, cardiovascular issues, dysautonomia is sleep disturbance, etc. What it isn’t, is it’s you’re not just flexible, it’s not harmless because it’s just pain. Joint hypermobile conditions are not always visible because the people avoid making the movement that makes him socially weird. Not something that everybody experiences not I can’t do this woods. Never could even when I was a kid. So it’s not something that you can develop or that everyone experiences and it’s not always cause for concern, especially if there are no other symptoms. So that’s what it isn’t. This is what it is.
31:55
Okay. Now Marfan
32:01
is interesting because it’s a genetic disorder that affects connective tissue caused a completely different mutation that tells the body how to make fibril and it increases a protein called TGF beta, the increase in T transforming growth factor beta, whatever that is, causes problems and connective tissue throughout the body, which in turn creates the fate and features and medical problems associated with Marfan. I’m just reading that slide to you because that’s what I knew about Marfan except the fact that it’s genetic, and you see patients that come in with hands that look like this,
32:43
who can do that.
32:46
But my Marfan is associated with mitral valve prolapse, aortic dissection, which is doesn’t usually turn out well. retinal detachment oops Fiber One, one arachnoid Dactyl II, which just means spider like extremities, extra long arms, extra long fingers, they’re nearsighted because of the connective tissue in their eyes, and they often have scoliosis. And it’s genetics. So dad has more fans. It’s right, it is recessive, it looks like so mom doesn’t have microphones. And out of the four kids just do the Punnett square. Remember when they made you take genetics and you learned how to do these in the major crazy. Okay, so, too, if they have four children, or one child, there’s two, there’s a 50% chance that if Dad had more fans, too, there’s a 50% chance that that child will have more fans. Does that make it dominant or
33:54
recessive? I guess it makes it. I don’t know. I’ll let you guys figure that out. But it is difficult but not always disabling. This was interesting, Michael Phelps, gold medalist swimmer built the muscles protected the joints and won. What was that? 567 Olympic medals. Basketball players, great actor Vincent ever I don’t
34:26
even didn’t even know his name, but everybody knows this guy. Howard Stern, Abraham Lincoln. And then you look up more friends on the internet. And that’s
34:39
the worst case scenario. So
34:44
Ehlers Danlos is female predominant, looks like after the age of nine. So in early childhood, you got about a 5050 chance of getting it diagnosed.
34:59
There’s more males right. So this is the incidence where it shows up. Right. But look, some people
35:12
are age 60 before it’s diagnosed, that’s not and if you miss the diagnosis, don’t feel bad. So I feel less bad after feeling seeing the slide about the patient same as 20 years ago.
35:27
So latency of time between the first symptoms and the diagnosis. And 636 patients. So age 4535, have a look that there are some people that aren’t diagnosed until they’re 50 or 60. That’s interesting. And distribution, lots
35:56
of pain in the lower legs, upper limb, spine, other joints.
36:01
Now this is interesting. And this goes along
36:07
with if you’re a psychiatrist or a GP, or a psychologist, or if you’re treating fibromyalgia patients and the incidence of body pain makes them show up as fibromyalgia or neck injury or low back injury patients. But they have this psychological profile that is characteristic of Ehlers Danlos anxiety, depression, eating disorders and that relates to the vagus we’ll talk about that. Autonomic dysregulation which is another word for pots or pots like syndromes, sensitivity to external stimuli. So everything jacks up their sensory cortex and high emotional reactivity. So the event is this vague and their reaction is that big. So you can download this paper and Ehlers danlos.com Or you can find it on PubMed. The American Journal of Medical Genetics and published in 2017. So, this is the paper
37:15
and it is fascinating that
37:21
I’ve had patients who are that come in and say yes, I’m bipolar. I have anxiety and depression and oh, yeah, my blood pressure’s kind of wonky. And they’re on medication for the emotions, but they’re 55 years old and nobody has diagnosed Ehlers Danlos.
37:42
That was interesting. So the neuro connective tissue phenotype, right, how they show up. They have anxiety and joint laxity. Okay.
38:00
But that creates behavioral symptoms, psychopathology as we would call it. So, increased internalization, decreased proprioception, anticipatory anticipatory anxiety. There’s some part of your brain that knows that when you do this thing that everybody can do. It’s going to hurt you. Your vagus is in charge of telling your brain this thing is going to create tissue fragments and the hippocampus remember the Vegas workshop. The hippocampus gets paid to remember things that hurt you. So the vagus, the hippocampus, the amygdala, that anxiety, phobias, somatic illnesses, dysautonomia blue Scarah sclera, easy, bruising. Eczema. dyskinesia means you don’t know where your body is in space. The kids that say, I’m just clumsy, dislocations. prolapses heterotrophic scars are those ones where there’s the scar is indented. So normal scars there’s a raised lump on the top heterotrophic scars are where the
39:25
the skin there’s there’s a ditch where the scar is.
39:31
defense mechanisms identified. At the extreme of continuous access fight or flight avoidance and dependency. So those are behavioral. somatosensory includes increased olfactory sensitivity. If your vagus is continually off your midbrain is continually jacked up. The midbrain is connected is sort of all information that goes from
40:00
your frontal lobe
40:04
to your sensory cortex. passes through the amygdala and hippocampus is how you know that Skunks are not nice, and gas is not nice. You smell that it goes through your amygdala and hippocampus before it gets to the sensory cortex. So there’s an emotional learning and evaluation that happens in between the time you smell it and the time you get to where your sensory cortex knows what that smell is. It passes your your hippocampus and amygdala evaluate the smell before it goes to your sensory cortex. Eye contact difficulty once again that’s an avoidance thing. Selective photophobia dismay. difficulty taking a deep breath that’s Vegas dysphasia trouble swallowing, choking palpitations, joint pain and enhance sensitivity to weather and chemicals. And there’s an article in Townsend letter we should have on the website
41:11
fairly shortly. If it’s not
41:14
already there. No, not there. I sent it to Kevin but he’s been really busy. And actually I’m not sure if I sent it but this enhanced sensitivity to weather and chemicals is related
41:27
to this
41:30
connection between the vagus, the amygdala, the hippocampus, and the sensory cortex and the cortex. Everything goes through the amygdala and the hippocampus and all that information. So the the weather the vagus has stretch receptors in the blood vessels that go to the back of your skull and in the dura and so when the barometric pressure goes down there’s less pressure holding those blood vessels contracted, if that makes sense, especially if the connective tissue basement membranes in the body in the body of the blood vessel
42:10
is not normal. Not strong enough. And barometric pressure
42:19
goes down the blood vessel stretches because the basement membranes not that strong. And the vagus goes Whoa, this is a problem. And the amygdala and the hippocampus go I remember this. This always gives us oops,
42:33
headache. So when the barometric pressure changes, the vagus sends stretch receptor responses to from the blood vessels and the dura to the hippocampus and the amygdala. And it all functions together.
42:58
And that’s why they’re sensitive to weather and chemicals. Isn’t that cool? So this is great slide go back and check on that. So the biology is this. So the normal connective tissue looks like this. Ehlers Danlos looks like this. It’s just not made right.
43:17
This is how the
43:22
how connective tissue is made. That amino acids are put together they make tropa collagen. Those make fibrils and the fireballs make fibers. So nanometers. 300 nanometers. fibrils are one micrometer, and the fibers are 10s micrometers. So these are really tiny. Structures. You put them all together and they make your TfL that holds your knee together. And it makes the ACL and PCL and your shoulder labrum and the ligaments and all that
43:59
that’s amazing. Structure function.
44:04
So this is how those are put together. I don’t know enough about it to even give a reasonable explanation. But this is what I found on the internet. So that’s that’s what I put on the slide. Some of you probably know more about this than I do.
44:26
Now the genes that go wrong look at this cut collagen gene is up here. It makes right goes to the writer zones puts together the pro collagen chain. These genes can go wrong, that gene can go wrong. So this is the
44:48
mutation affects structure synthesis and stability and certain types of Ehlers Danlos EDS for EDS six if it doesn’t hydroxylase
45:01
or make the right disulfide bonds, eds seven defective pro collagen
45:16
collagen fibrils
45:16
selfless assembly. So the This is why there are so many genes involved in airless danlos. Look how complicated it is. And any one of these genes can go haywire and I guess in a less technologically advanced or less kind society, they want to died out as it is. The recessive genes especially will reproduce and we keep the child alive but it has certain disabilities that you saw and the other section where the problem happens. So osteogenesis imperfecta scurvy, this is why so this hydroxylation is done by vitamin C. Right? collagen fibers anyway. So where the problems happen. Now what does the vagus nerve have to do with Ehlers Danlos so the vagus nerve affects everything and keeps anxiety and depression at bay opposes the sympathetic response to stress conveys information from the organs to the brain. It’s not just the organs 80% of the information that goes from your body to the brain goes by way of the Vegas. The Vegas workshop is going to be even bigger this year than it was last year controls the throat muscles to send food and air down the right tubes. insulin secretion and glucose in the liver suppresses inflammation by the cholinergic anti inflammatory pathway. Fainting when over stimulated leaving leading to temporary loss of consciousness and bladder control.
47:10
Controls digestion
47:13
is responsible for feeling full increases gastric juices, gut motility, stomach acid and actually determines there. There are sensory receptors in the small intestine that determine what nutrients you’re getting tells the brain Hey, you should really start craving pickles scuze me? Well, it’s because you need something in the acid or the contents of the pickles. And that comes out of the vagus sensory receptors in the small intestine lowers blood pressure. In the case of illness, emotional state decreases heart rate, vascular tone. It controls every muscle involved in speech. So patients with
48:05
vocal cord dysphonia spastic spasmodic dysphonia gag reflex swallowing coughing so this is all of the vagus reflex
48:16
So this slide just didn’t have the precision but look at this. Ehlers Danlos, Upper Cervical instability cervicogenic nucleus trigeminal neuralgia, right. Look at these arrows. It’s you can I don’t know maybe make the picture smaller look it up.
48:37
vagus, nerve anxiety autoimmune so these are all interconnected. Vertigo. eustachian to Menieres right cervical sympathetic cervical spinal cord, stimulator and instrument insufficiency decrease Look at this. It’s just just Sam. So these are common clinical
49:10
observations and peril patients with Ehlers Danlos and then look at the vagus nerve and our EDS patient. What controls gastrointestinal issues the vagus Potts has to do with the vagus right orthostatic tachycardia, the vagus hypotension the vagus cardiogenic synthese syncope, the vagus nerve significant arthritic arrhythmias means it’s not the cardiac nodules that are problem, right. All of this is the vagus so how does it work? The vagal afferents notify the brain limbic centers of infection and trauma, pathogen associated molecular patterns damage associated molecular patterns, tissue fragments, the midbrain limbic sector centers so in our world 89, up regulate, they they get jacked up. They suppress the vagus and during stress, injury, infection threat. The vagus is suppressed because it gets in the way of survival, tissue injury is constant with EDS. There are constantly
50:25
calm constant, Dam’s
50:28
damage associated molecular patterns that are associated with their Ehlers Danlos. Now, this makes me wonder about the acquired Ehlers Danlos that comes with infection. Does that have to do with the
50:43
power amps? So that’s the pathway. So how does the vagus work to regulate the immune system? Well, the sacral nerves go to the colon. The vagus nerve goes to the spleen and regulates T cells and macro and macrophages,
51:08
celiac gland gland, those go to the colon and all of this feeds back into the immune system. It is an immune the vagus is immune, regulatory. So how does airless danlos affect the vagus? Well, the damps that come from the tissue injuries suppress the vagus. But my theory, look at the branches the vagus is everywhere because of its connections in the blood vessels. There’s no place in your body that doesn’t hear doesn’t communicate with the vagus because everything’s attached to everything. But I think in addition to the dams, the vagus is attached to everything in your small intestine, look at that. And every time there’s pain in the stomach, so every time this patient stands up, the, I don’t know 20 pounds of liquid and food and stool that’s in the digestive system
52:12
pulls on the intestines, and that pulls on the vagus.
52:19
So it’s connected physically, structurally to the vagus every time the patient stands up. The connections cause traction imagery to the vagus. And impair vagal function.
52:30
Go back to the
52:31
psychological aspects anxiety, depression, eating disorders, autonomic dysregulation, sensitivity to external stimuli, high emotional reactivity, all are associated with limbic system. activation, and that’s associated with the vagus and that’s associated with the membrane. You see where I’m headed with this right? All right,
52:55
so how do we treat it FSM treatment.
52:59
So the first thing I did on the first airless danlos patient I saw was I treated 124 and 77 on her for an hour. Alternating I think, and I did it actually on her. Her major symptoms that she told me about were just the fact that she couldn’t she couldn’t hold a water bottle. She could not do that.
53:29
So she couldn’t do that. Her hand muscles wouldn’t work.
53:35
So I ran it from neck to hands and in 60 minutes, she could pick up a water bottle that was painful. Then she came to a seminar, two, three months later, and I looked at her pain diagram. So I ran 124 and 77 with contacts at her neck and feet. And that worked. I think it was between an hour and an hour and two hours it was after, at the end of the day at a seminar if you weren’t in Denver a couple years ago, I think it was 2018. So this, this particular 124 and 77. Reduced hypermobility in every hyper mobile and EDS patients that I’ve treated and there’s probably
54:19
10 In the last two years. Finger range
54:24
of motion returns to normal. That was kind of interesting there used to bending to 110 with the index finger and all of a sudden it stops at 80 I was weird. Elbows and Knees were not as responsive. So at the end of 60 or 90 minutes, elbows and knees were still hyper bendy skin laxity was reduced. That was kind of interesting. And that brings me to I keep saying we can put tissue back this not there. What are we doing when we run 124 and 77? I have no idea. None. Anybody gives me a Dinah cross if you’re listening. If you come up with a model for how it is that 124 and 77 works. I’m with you. That’ll be fun. And the effects last about a week. And male patients typically don’t retreat themselves. Yeah, right. I’ve lived this this. I’m 28 years old. 30 years old. I’m not going to treat myself. female patients tend to treat themselves so that was interesting. So 124 is what you treat with a connective tissue but that doesn’t change the pain diagram. When I saw the pain diagram in the earliest danlos patient, it looked like cervical trauma fiber patient circles that shoulders, elbows and knees. The outside world the earliest downloads world thinks that this
55:53
comes from the Giants. It went away with 40 intent,
55:59
the body pain that she had went away with 40 intents. So think about this pain diagrams. are the same. Well, what’s the disc annulus made of connective tissue.
56:11
If the connective
56:12
tissue doesn’t work, right, what’s going to happen to the disc annulus it’s gonna have little cracks in it, it’s gonna leak. Right? The nucleus material can then affect the spinal cord just same way that cervical trauma fiber does. So people are saying the pain is from the joints, but in our world, it’s 40 and 10. You look at that pain diagram. And if I did my job, right, you’re all thinking about fibromyalgia. So you’ll treat these people for 40 intend to get rid of the pain, but you may or may not think of treating the connective tissue. So from now on, every time you see a fibromyalgia patient, ask them to bend their fingers can they touch the wrist with their thumb? I don’t do the other parts of the Baden scale but those are easy you can do those with them sitting in a chair. Then 100% of the time you treat the vagus how do we treat the vagus well quiet the activity of the medulla where the vagus comes from and the midbrain, which turns the vagus off and then treat the vagus for trauma. If it’s getting traction injuries. It’s definitely traumatized increased secretions and vitality and the vagus. And the ability to treat the vagus in 60 minutes makes FSM unique. That helps reduce psychological issues temporarily improves digestion. They get up off the table they don’t have pots anymore, reduces inflammation which can be responsible for the joint pain. And you put the context of the neck and abdomen because technically the vagus stops at the abdomen but I guess you could run it neck to feed. Once again. This goes 60 minutes now in the Vegas workshop. All of these run for four minutes. The other night I treated an airless danlos patient
58:07
for 90 minutes
58:10
and 30 minutes of that I ran increased secretions in the Vegas because it made her so stoned she couldn’t talk and I just anytime I went off of it. She got on stone so I stayed on it. Her heart rate stayed the same didn’t change
58:27
so I’m just saying then the concussion in Vegas you have a
58:34
concussion in Vegas. It’s the perfect combination contacts at neck and abdomen. So remember 6.8 changes genetic factors
58:42
doesn’t really maybe. So
58:48
the basic concussion protocol 6.8 and 38. But then you still need to do 1494 40 and 89. Now the question is 40 and 92 because these people are hyper sensory aware, not sure if this would help. Why would you want to quiet down the sensory cortex? Because this midbrain, the amygdala and the hippocampus have been screaming at it for 27 years. And so their sensory cortex is hyper aware because the amygdala and hippocampus type of
59:28
burn. Sensing
59:33
threat no matter what the patient does, and then trauma in the vagus increased secretions. This says four minutes each. I’ve had one experience where around at one on one or nine for 30 minutes patients just kept feeling better. It was interesting. Now after you get rid of the tendinopathies so you may have to treat 120 foreign one I may one as well as 124 and 77. These are competitors slides. So from the from module to the pain in module one the pain and injury right so 41 through 96 reduce the nerve pain, then treat if these tissues have been chronically injured, are you going to have adhesions between the nerve and the fascia? So even if you get rid of the error stainless on the first day, three days later, you’re going to have to treat scarring between the nerve and the fascia because you’re not going to do that the first session release the adhesions move the nerve and fascia remember that you move really slowly.
1:00:40
And then what do you need to do? Or cerebellum has
1:00:43
no idea where this arm is in space. Right because in 60 minutes you just changed everything. So increase secretions reconnected to the brain, increase secretions in the nerve, the spinal cord, follow, follow the spark ad for the cerebellum, and this one is where I spend most of your time because we’re not sure if you want to increase secretions in the sensory cortex or decrease them. That one I haven’t played with. But I did increase secretions in the cerebellum with the context of the spine and the nerve and you can do this from neck to feet you can do it from neck to hand. We had pictures from active and but you could run this neck to feet and have the patients stand up and just do a side to side weight shift and then move their hands and then move the affected muscles. So you can see this patient three times in the first week. Run connect 124 and 77 for a while and then do this part, like on the second or third visit and that week. And then so you see how it kind of put it together into a treatment plan. And then the digestive issues you’re gonna have to treat small intestine or SIBO. And
1:02:17
so, trauma
1:02:19
and vitality in the parasympathetics You got to be careful if it’s the diarrhea kind of SIBO treat the infection. Why would there be infection? Well, because it’s SIBO and they have dysbiosis and bloating every time they eat. That’s their definition of digestive problems. But in our world what is it? Well the vagus regulates the pancreas, digestive enzymes, the small bowel and the colon. So you can I mean this is an hour’s worth of treatment even on accustomed care. So you
1:02:53
can take SIBO and the vagus to it. So you treat the parasympathetics and include the vagus and then treat I would treat just Yeah, I generally with SIBO I just treat the small intestine. But if
1:03:22
enzyme secretions are a problem, you know this we’re not dealing with the eyelids. We’re dealing with the pancreas and digestive enzymes. So that’s the thought. So remember this from the core disconfirmed set pain patterns are pretty much identical and you can’t know the sides of reverse but can tell the difference. Said pain is worse with extension. Disc pain is worth with flexion because these patients are hyper mobile at every joint, they’re going to be worse with flexion and extension. So you’re going to treat facettes and Dennis, how do you do that? I would do sub acute facettes and sub acute discs. I’d modify it to take out 91 Right. It’s not a one visit fix this is going to be the part of their pain that gets bad every time. You have to strengthen and stabilize spinal muscles. patient will need to treat themselves regularly at home these people will end up with custom cares, and then see a good physical therapist that knows how to stabilize the spine. And that’s just a setup for neck
1:04:31
and low back extremity joints. Create a
1:04:36
protocol I don’t have an EDS protocol and Customer Care. And I gotta tell you, I’m not going to make one you can do this. Modified I would copy clone extremity joint MLT b sub acute, it’s not acute. It’s not chronic. You don’t want to do chronic and just modified shorten it. What is the problem? What gets torn and broken the connective tissue right the ligaments Yep, the joint capsule. For 8214 as meniscus if you haven’t been to the advanced, this is new for you. And 191 So tendon so you get the flat tendons around tendons and pretty much everything made of connective tissue in the joint. Then when the joint bashes around what gets damage, inflammation, what’s in the joint or tissue as cartilage gets beat up periosteum because the connective tissue and the tendons and the ligaments attached to the periosteum so get it gets inflamed it might even get torn and broken when it wants to tendon 190 fives the bursa one hundreds of ligaments, connective tissue 5939 49 Then vitality and all those parts. So you could create an EDS joint protocol and EDS for set and disk protocol.
1:06:05
That’s pretty much though.
1:06:06
You can do whatever you want. It’s not like we know what we’re doing with this one. So so far I’ve seen 10 patients in the last two years. The results are normalized range of motion. Like in 60 minutes in my brain. I don’t understand it. Reduced body and pain is 40 and 10. That’s easy. reduced anxiety and depression. Their mood changes when they get off the table and it doesn’t hurt. They don’t know what to do with it. Not sure why so the vagus are just you know we’ve treated the vagus or affected the vagus or is it just that you’re less anxious and depressed when your body pain is in a seven and they improve digestion. Results last about one week. No patient has been followed long term and there’s no negative outcomes or side effects. The thing I forgot I just remembered what I forgot to
1:06:58
put in or is it something in the spine? This is placed where I guess I need to put it in
1:07:11
one of the Ehlers Danlos patients I treated 124 and 77 did great everyplace else. But in his neck it was 124 and 443. The Dura so the dura is made of connective tissue pretty dense connective tissue, so it’s torn and broken. And the dura that’s that’s the thing.
1:07:33
All right. So what don’t we now well, you’re just
1:07:40
there’s so many things we don’t know because I only have 10 patients. Once you get to 50 or 100 patients in case reports either stuff I do or stuff you do. Can we help with the vascular effects? Connective tissue, treat the arteries. Treat the capillaries, right? Trauma, torn, unbroken inflammation, vitality Can you change the genetics? Remember 528 The solfeggio frequency to repair DNA
1:08:10
in the connective tissue, baby 6.8 and 38 We don’t know who it’s not going to work on. The more severe forms obviously we don’t see them much.
1:08:24
The hypermobility syndromes we see all the time, just miss it. We’re not going to miss it now. Can you modify the treatments so they last longer? We don’t know that. Patients need a custom care. So what do we need as a group everybody that’s on this?
1:08:42
More cases?
1:08:43
Obviously I need you guys to start recognizing it and treating it. More experience with what works and what doesn’t work. How to how do we know so when our patients can have realistic expectations?
1:09:02
You can’t say to somebody piece of cake when I kind of do that, but
1:09:07
I really don’t know what 10 cases when you get to 100 Then you come to know I need more long term data. I need any data. I love treating but I’m terrible at collecting data. I don’t do the range of motion. I don’t have the baton score and I should but the first 10 cases, you don’t expect to be able to treat it so you don’t really check right. Simple hypermobility is fairly common and musculoskeletal patients but EDS is rare in the general population. And the good news for us is nothing else works very well in these patients. So we have really a chance to as a group to make a huge difference in these patients. lives. So the symbol of the Ehlers Danlos.
1:09:53
Website is, you know, when you hear her
1:09:59
her feed, so for horses, not zebras, while the EDS patients are zebras, they come with a constellation of symptoms that nobody recognizes so that’s what we know what we need to do what you can do. Go back and read this over again. The advanced this year is live stream online, which is good news and bad news. I’m gonna miss seeing you guys and I’m going to miss the great food at that at the hotel in Phoenix. But we’re going to do a five day live stream, which means I’m going to be recording a five day we’re not going to use an old one because as you’ll hear from Kevin I was changing slides at
1:10:47
two o’clock this afternoon. So the
1:10:50
the core will probably be slightly different David Mazanik can’t help himself. He puts in new slides. The FSM sports was supposed to be in Phoenix but Kim Pettis is holding it January 9 and 10th you can find the directions for how to sign up February 25 and 26 as
1:11:11
the advanced so my problem has been multitasking There we go. I don’t know it’s a little arrow there we go. Okay. copy of the slides. Yes. Oh, so
1:11:35
activation MCs that’s gonna just that’s going to be in the five day core or the neural visceral core module. David Masnick does a great job with MCA as it is always associated with the vagus. The reason for the MCS is not part of the dysautonomia. It’s because the vagus has its job to suppress her regulate control the immune system go back and look at the Vegas workshop from the 2020.
1:12:16
Are we there? Can you hear me?
Bill Clearfield 1:12:17
Yeah, we’re here with Carol.
1:12:19
Hi there.
Bill Clearfield 1:12:20
So everybody, this is Carolyn like making she was our speaker for the evening and I guess the rest of the webinar was announcements and right for the cut it off there. A lot of questions. First of all, what is FSM? Yes, that because a lot of our people are not familiar with it. Right? And what and, you know, you always tell tell, tell the story. So go ahead. Go ahead and we’ll take it from there.
1:12:56
Okay. Well, a lot of those numbers that you saw our frequencies from that I got from an osteopath who bought a practice in 1946 that came with a machine that was built in 1922. So in the US from about 1905 through 1925.
1:13:27
There were people
1:13:32
EMD is an osteopath used frequencies to treat patients and electromedical digest recorded the data they had journals, they had research clubs, they shared frequencies. And when the Flexner Report came out, by 1927 or 28, the use of frequencies homeopathy, herbs, nutrition was all illegal. So drugs and surgery was the only thing that was left. And the machines either went on the junk heap or went in the back room, and Harry van Gelder was an osteopath who bought this practice and 46 walked in the background lifted up the sheet, and there was this machine and a list of frequencies and he taught himself how to use it. GEORGE Douglas went and worked with van Gelder in 1983. He and I got together in 91 and we started using the frequencies on a microcurrent machine in 1995. And my practice in Portland, I’m a chiropractor. I do physical medicine, and we started out treating muscle pain then went to nerve pain then I lectured at the National Institutes of Health on one frequency combination that treats the full body pain associated with fibromyalgia 40 hertz on Channel A is the frequency to reduce inflammation 10 hertz on Channel B is the frequency to address the spinal cord. I treated 54 patients with 54 patients with fibromyalgia from spine trauma
1:15:32
and
1:15:35
58% of them almost 60% of them recovered from fibromyalgia in four months, but 100% of them had their pain go from an average of a 7.4 to one 1.4 in 60 minutes. So I took these cases to NIH in 2000 and gave a grand rounds presentation at the invitation of Jay Shaw. And I said okay, I’ve done this
1:16:04
at that point
1:16:05
27 times, there are no failures. There are no side effects. Somebody needs to tell me how to measure what it is that’s happening, or nobody’s going to believe it. So Terry Phillips came up and he said, you send me a spot of blood on blotter paper and I will tell you what’s changing I treated a patient sent him the blotter paper. And six weeks later, five weeks later, he sent me back data that says that 40 hertz on a and 10 hertz on B reduces all of the inflammatory cytokines interleukin 168, TNF alpha CGRP and substance P at logarithmic rates by factors of 10 and 20 times that’s not possible then substance P is produced in the spinal cord. And that’s when we found out that substance P went down from 88 to eight it went down by 10 times and that’s when we knew that 10 hertz actually
1:17:22
address the spinal cord.
1:17:23
So those numbers that you saw that webinar was recorded for my practitioners when I first started with Ehlers Danlos at this point, we haven’t had any failures and it’s in wide use across the country. They go from and I do keep data now. They go from a Beighton nine to a Baden eight, or one. So some of the elders danlos patient I know there’s one person in the group that isn’t Ehlers Danlos patient. The last of the Beighton score than nine is they can bend forward and put their hands flat on the floor. And when you shorten the connective tissue, certain proportion of Ehlers Danlos patients have tethered cord so they can still want you to tighten the connective tissue. The tethered cord gets worse than we treat the Tethered cord and then they can’t touch the floor. I have one patient She just left. I’m still in my office at six o’clock on the West Coast. She just left. I tweeted her three times six months ago and she is still a Baten Zero out of nine. She can’t touch the floor. Her little finger stops it’s 70 degrees. It’s permanent
1:18:54
on her.
1:18:58
So the frequencies we did animal research in Sydney, Australia and found that the frequency to reduce inflammation and the spinal cord sorry, reduce inflammation in the immune system, reduced lipoxygenase mediated inflammation by 62% and four minutes. And I sent Dr. Clearfield the slides for the FSM introduction and perhaps he can send them out to people that are listening. We’ll put it on our website. Yeah, we put them on the website because the data is just ridiculous. So locks inflammation by 62% in four minutes and that was with everybody blinded talk talks mediated inflammation went down by 30%, which doesn’t sound that great, but it’s equivalent to injectable toradol when toradol was tested in the same animal model by the same researcher and injectable toradol is about as good as it gets. We have published papers on head and neck pain myofascial pain, low back pain another large collected, controlled trial from India on low back pain torticollis nerve pain, scarring and nerves. I think there’s about 15 Peer Reviewed published papers at this point. The book that tells you how FSM was developed is called the resonance effect. And that is it’s an audio book and and it’s at Amazon. It’s a quick read. It’s the story of how I developed it. There’s an FSM textbook that was published by Elsevier and that’s a little bit outdated, but it’s Ehlers Danlos it’s amazing. The number of people that have it and don’t know. So everybody now that comes in I treat chronic pain patients in this clinic and everybody that comes in that had an auto accident 15 years ago or 12 years ago, and they still have neck pain. I do a Beighton score on every new patient just about so
Bill Clearfield 1:21:35
because it’s easy, right? Yeah, really. Are you allowed to talk about your most famous football player? Is that is that Oh, yeah. Well, he
1:21:45
he mentioned me so that was fine when he was I treated Terrell Owens after he broke his ankle. He had a high ankle sprain which means that the interosseous membrane between the tibia and the fibula was was shredded. Hip valse, the deltoid ligament. Hit an open spiral. fracture on the fibula, and I treated him. I flew out treating him as soon as he got out of surgery. They started treating the fracture right after he was injured on Sunday. I treated him Tuesday, and they said he wanted to play in the Super Bowl. Six weeks after the surgery. And the surgeon was running around saying that it was a career ending injury. Anybody with a microphone and we had him the spiral fracture was healed. The ligaments were stable in four weeks and when he got to Jacksonville for the Super Bowl, he couldn’t run on Tuesday because everything cramped up. I arrived on Wednesday and we started we spent five hours Wednesday night taking apart the scar tissues so the frequencies to dissolve scar tissue are extraordinarily effective. FSM has basically applied biophysics so our model for how it is the frequencies work is that the frequencies appear to change cell signaling and change what happens inside the cell to change how the cell has an output and with scar tissue. The physics of it is that scar tissue is like a rubber band that’s wound on itself. And the frequencies appear to resonate with the bonds that hold the scar tissue in a shortened configuration sort of liquefies them. And when the bonds break, they’re not they don’t connect back together. So we treated him for five hours on Wednesday, two hours on Thursday. He played in the Super Bowl like he’d never been hurt and when he got the went into the hall of fame this year he named me and Chelsea and Brian as an buddy Prem has trainer as the Dream Team. And then Maura has a question combination therapy she find helpful to use with it.
1:24:32
You have to have a stable state is what you’d call it. So
1:24:39
with let’s say Ehlers Danlos since that’s today’s topic, use FSM. The patient still has SIBO or irritable bowel. They have to make connective tissue so they have to be taking vitamin C. They have to be on a hypoallergenic diet at least until we get the vagus nerve working and the food sensitivities to quiet down. Almost anything we treat. So for example, if I’m treating a
1:25:19
nerve pain or disc bulge,
1:25:24
you have to take essential fatty acids to get the inflammation down. Right. So use FSM and you have the patient do certain exercises to stabilize the spine around the disk. Bring circulation to repair the desk. Treat the nerve nerve pain is easy. I have a young man who left here today. Last August he was hit by a car while on his bicycle. He fractured his femur. They put a rod down the femur without putting a screen to keep the fat embolized down. So he was in a coma for eight and a half days with an embolic stroke. Some of the fat and lie are around the thalamus. So he has thalamic pain syndrome. On all the drugs that they could give him. His pain level was between a seven and a 960 minutes on the frequency to quiet the activity of the thalamus
1:26:35
and
1:26:38
he’s out of pain. So he left here last week with a what we call a custom care. It’s a small unit. And he came in today with his pain, about a six because he’s moving and then I just put him on the unit and he can go to bed at night and have his pain at a zero when he wakes up. That’s extraordinary. That’s impossible. So the website, so I love osteopaths and I love having people that are good at and interested in a combination of Physical Medicine and integrative medicine or functional medicine. Take our course because you guys know what to do with it. So when you can treat nerve pain, that’s easy when Fibromyalgia becomes the easiest thing you treat when Ehlers Danlos is, at this point, hundreds of cases into it by report. Nobody’s published anything. It’s a slam dunk. I haven’t had any failures. And we found out that probably 20% of every class that I’ve taught in the last year is Ehlers Danlos patients. There’s hypermobile patients everywhere. So go to frequencies specific.com and
1:28:11
find out how to take a course
1:28:15
you can take it on video which I recommend because it’s five days long because I don’t know what to send you home without the visceral section is the last part. Physical Medicine is the first four and a half days and then everything from asthma to irritable bowel to brain injuries to concussion to vagal tone to kidney stone pain. There’s one frequency combination the only thing it’s good for is kidney stone. Pain takes the pain away dissolving the stone is a different project. So is there anything else?
Bill Clearfield 1:28:51
Well, there’s some questions in the chat and then if folks want to hang around, so my my new practice partner Jen boscovich is one on one on the on the webinar tonight. And as luck would have it, she’s our Ehlers Danlos patient so Oh, for heaven’s sakes, like to hear, you know, a little bit from you know, right from the horse’s mouth sort of. She She agreed to, you know, talk and answer some questions. So but there’s a couple of questions here. Now, some of it is about healers. Danlos Syndrome, not specifically about what you do so like is Ehlers Danlos patients more prone to mold for instance? That’s a question.
1:29:34
Well, it’s the challenge is that the vagus nerve is turned off by others danlos because it’s turned off or down by infection, stress and trauma. And when you have fragments of connective tissue floating around the vagus nerve is downregulated which I don’t know that it makes you more susceptible to mold but it’s it’s a contributing factor. Somebody said more, where can we find practitioners if you go to frequencies specific.com There’s a tab that says Find a practitioner, and I’m so excited because our new new looking website is being launched on June 14. So that’s pretty exciting. But there are FSM practitioners, 5000 practitioners and 23 countries at this point. So there’s about 2500 In the US, and you just Google the we charge to be on the list. So not everybody is chooses to be listed. But it’s $89 a year. And what I’m hoping is that you and your team will take an FSM course on video. The equipment is an expensive, it’s produced in the US it has 510 K It’s ISO certified. And it has a CE mark.
Bill Clearfield 1:30:55
I’m getting close, right. I’ve threatened for two years now.
1:30:58
So it’s about time though.
Bill Clearfield 1:31:00
Hi now. So I’ve actually spoken at the last two advanced courses for FSM Carol has been gracious to invite me and I’ve been speaking about traumatic brain injuries. And I even did a little little homework and looked up the frequencies and all that one so
1:31:22
well and we have you come and speak because we treat brain injuries and strokes and the end. And I the endocrinology and the nutrition that it takes to repair the brain is beyond my scope. And it’s the missing piece is the endocrinology of it so I’m really we’ve really enjoyed having you there and your lectures are really, really popular.
Bill Clearfield 1:31:51
We try to have fun and your group was really terrific to be with to there are really a fun group. So
1:31:56
yeah.
1:31:57
So Dr. William Can I just have two minutes. Just to interject here because your speaker is very interesting. I’m Dr. Hi, Alyssa and I enjoy everything you present. What is what I think it’s missing? is also the redox medicine because once you understand redox medicine makes sense everything that you say and I think airless Danlos Syndrome, there is mutation in the genes that makes the collagen corrupted, not perfect. And then you end up with this hyper rusticity and there’s two way to manage it. There is a way that you can override the mutated genes. And that is a big big topic that we’re going to talk about it throw a drug is called I’m legs to an axe, but also there is an anti inflammatory part of it and oxidative stress which you are taking care of it by your microcurrent electrotherapy. And I can see that those electrons they are going to reduce those free radicals and they will dumped the oxidative stress and the redox potential. And once you’ve dumped the redox potential in the macrophages, you will definitely decrease all the cytokines profile because what activate No
1:33:16
wait, stop weight. It’s not just the current. If you don’t use the right frequency, it doesn’t work. So when we treat the Ehlers Danlos there is one specific frequency combination that normalizes range of motion and there is one specific frequency combination only 40 hertz on a and 10 hertz on Channel B that gets rid of the body pain by reducing inflammation in the in the spinal cord. So the electrons do theoretically, increase ATP production. But it’s not just it is absolutely not a current effect. The frequency has to be correct for what the patient has. So I understand what you’re saying about redox potential and current. But it’s it is a very frequencies specific effect that is supported by data.
1:34:22
So I’m not hitting that the frequency it’s part of delivering electrons in the way that it will reduce the redox potential you need to have that voltage and you need to have that delivery and the right amount. Otherwise it’s going to be it’s actually may turn and become oxidative but that’s not you know, the frequency I’m not negating the frequency. I think frequency is part of it. But what I’m saying is that those electrons are neutralizing those free radicals. And part of it it decreases inflammation directly and other part what’s the same mechanism of grounding, same mechanism of electrotherapy fairground anybody
1:35:02
you want and you’re not going to get them from a Beighton nine to a Beighton zero and 60 minutes,
1:35:08
right the frequency of add more value to to deliver those electrons in more efficient way. I understand that I’m not negating that. Because you said putting the microcurrent in the neck part of it is stimulating the vagus nerve which actually has into anti inflammatory arm of the autonomic nervous system. So that’s part of that, Carol,
Bill Clearfield 1:35:30
so yeah, so is EDS, an auto immune disease?
1:35:34
No, it’s a genetic condition where the connective tissue is made incorrectly and that’s one of the reasons I don’t understand how it is that I’ve treated this patient three times and she no longer has it. I don’t understand how it is we can treat them once and it lasts for seven days or two weeks. i How do you reverse How does and it’s very specific frequencies how those two frequencies reverse a genetic condition and change the cell signaling that creates connective tissue. So
Bill Clearfield 1:36:20
somebody smarter than me that like this person over here, this is Jen. She’s our new practice partner actually again, and unfortunately has Ehlers Danlos Syndrome. So it’s which I don’t think I’d never heard of before till she mentioned that. I had to go look. It’s
1:36:42
i I can’t even tell you. If no. Yeah,
Bill Clearfield 1:36:47
like just let her like talk a little bit about some of some of the her experiences. Sure. I know we had a hip out of place about two weeks ago and she just turned a certain way and boom,
1:36:59
yep, just dislocates the joint capsules are loose,
Bill Clearfield 1:37:02
and Joe put it back in place but yeah.
1:37:08
Yeah. Thank you, Carol for just bringing so much awareness to this. This is for someone like me, I’ve been in the medical profession my entire life, didn’t recognize it in my own family and took me way too many doctors and surgeries to realize, you know, to be diagnosed and I was still lucky enough to be diagnosed in my early 30s. But you know, what was fascinating was the diagnosis made me feel not crazy. Because no one can find answers, but we know that, you know, looking back at my life, I guess it’s like, wow, I was never really sick. It’s always been Ehlers Danlos everything I’ve ever had are problems. I haven’t had exploratory research on my stomach as a kid. There were so many just strange things. I had joints that would slide in and out of place was normal for bladder control even for my whole life has been, you know, normal headaches, all of that the chronic pain didn’t start until trauma like a car accident. So it was still as a kid so I’ve had it most of my life. I would love to try therapies and be trained. This is amazing. I can share one. There was one time in my life that I did become completely pain free. And I do want to share that part. I don’t know if that would help or not. But I was having so many digestive issues. It was getting so severe. I really didn’t want to eat I just didn’t want to eat anymore. And I knew I went to a Detox Spa and when about was like eight or nine days where we didn’t eat food at all just drink. Teas did a lot of detox colonics really focused on alkalizing the body they did not do IV therapies at that time, and a lot of focus on lymphatics and I left and I think I had two weeks and I was like wow, this is what normal people feel like I had. I had no clue people could feel like that. I was I was blown away. So I know that there’s some detox component and the more I can detox the better off feel.
1:39:23
And I Yeah,
1:39:24
well. The other thing about the full body pain, everybody thinks the joints hurt because the ligaments relax. And in point of fact, the the pain diagram for others die in downloads is the same as for fibromyalgia, from spine trauma. And in our world. You look at the disc annulus and it’s made of connective tissue and then Ehlers Danlos. The connective tissue is not made correctly. So the disc annulus is leaky. And the disc nucleus is the biologic equivalent of battery acid. So it activates thins actually the anterolateral pain pathways in the spinal cord and creates inflammation in the chord and that creates central sensitization, which puts everything
1:40:20
off. I’m trying to figure out a way that the
1:40:29
that the two week experience you have happened and I’m thinking about that, but there’s a question here who have Yeah, Beverly says, more vitamin C to make more connective tissue and then more. Do you think your therapy is turning off the malfunctioning jeans, a bill ability to express itself and that appears to be how FSM works is the protein receptors on the outside of the cell control the kinases control the genes the messenger RNA and the micro RNA that
1:41:08
control the cells output.
1:41:11
We think the frequencies affect cell signaling by affecting the protein receptors configuration because the effect is very, very frequency specific. So when you have Ehlers Danlos every time you stand up with food in your stomach, you basically get a nerve traction injury in the vagus nerve because the vagus is attached to your gut. So the vagal nerve goes down, inflammation goes up. Anxiety and depression go up. Digestion gets worse, and it’s this vicious cycle. So not eating anything for two weeks, might have might have just given your gut arrest so that you the vagus nerve would come back on and your inflammation would go down.
1:42:06
Does that make sense? Yeah, pretty much. Yeah.
Bill Clearfield 1:42:13
What was the aha moment when you when you found out that what what tests did you do that said, Okay, now, I’m Ehlers Danlos. And I’m not you know, just Michigan osis.
1:42:25
Well, I’ll tell you, I had, excuse me. I had a big surgery. I had rectus Thiele repair SR steel repair and a hysterectomy all at the same time. 31 And, yeah, so obviously not planned. That was my second little pumpkin. We’ve delivered everything. So after surgery surgery took a really long time and they said something’s not right. The tissue just kept stretching and stretching and stretching like nothing was was the surgeons were blown away in there. So that sent me finally to a geneticist. I had had so many surgeries already. Before that. unfortunate but I was so happy to land in Stanford. So I was in Stanford, it was their morphants clinic. They did all the testing that the genetic markers, but it felt, I’ll tell you just hearing that diagnosis. It made everything makes sense. And it was such a relief. Yeah. So that was the
1:43:35
Yeah. How many genes are involved in Ehlers? danlos. Today? No, it’s a constellation because there’s a big there’s a wide range of symptoms.
1:43:47
There’s a wide range of symptoms and they even then at the time, they told me, you know, I’ve been diagnosed for about a decade now. So they even told me there were still there was still a lot to go. There’s there’s more gene markers that are out there that they haven’t identified yet. So they said hey, even if the markers don’t come back, you obviously have a connective tissue disease. So
1:44:13
well done. Well, if you can find somebody, you guys are in Reno. I don’t know who we have in Reno but like literally, it’s, it’s it doesn’t even require manual therapy. You just sit in a chair and your pain goes down the vagus nerve starts working. The Beighton score like you become not stretchy which feels weird. And if you have any sort of tethered cord symptoms you have to treat those separately. So it can be two or three sessions or two or three machines in one session. But I don’t have to do anything physical until with you until afterwards, when we start reconditioning and strengthening the muscles. Once the neural the muscular attachment to the bone is normal tone. It’s not stretchy and get rid of the adhesions and the nerves and the nerve pain and it’s fairly straightforward these days. Like that. I think I did that webinar on what 21 That was two years ago. We’ve had classes now where 30 people in the class and we had eight of them with other 27 people in the class we were to a big treatment session with eight patients with Ehlers Danlos being treated after the class. And all eight of them got up pain free with normal range of motion.
Bill Clearfield 1:45:46
All right. All right, Jen, we know what our next what are our next academic assignments will be putting in the order tonight.
1:45:58
I will let you go. Thank you so much for having me.
Bill Clearfield 1:46:01
Thank you so much. There’s one more question here. Somebody asked. Can Hi TGF beta one not be more fans or is it always more fans so I don’t know if that’s your?
1:46:13
I haven’t seen that many Marfan. I haven’t seen any Marfan patients. Everything I’ve seen so far is Ehlers Danlos. But I don’t see why it wouldn’t work. It’s if it works in EDS. It should work in my fans. They’re not going to be they’re not going to get shorter. They’re, you know, their limbs are still going to be the same length but the connective tissue portion of it may resolve. We don’t know until we do it. I didn’t know we could treat Ehlers Danlos into light until I did it. And then I did it twice and then I did it 10 times and now we’ve done it about 50 or 60 times and so far, we haven’t had any failures. So we’ll find out. Let’s do
Bill Clearfield 1:46:59
one more question.
1:47:01
Yeah, just one question in your time, Carol. Excellent. And I appreciate everything you’ve done figuring this out, and I’m trying to learn it. Now. I have a patient 16 years 16 year old female, who after COVID A couple years ago she actually lost her ability to control her control MC duration and she has to ended up having to start self capping herself and also has an ostomy to to flush out her rectum and to help her defecate. And I actually suggested to the mother that we consider pulsed electromagnetic frequency only just out of principle. But I was unaware of this, of this strategy. Is this a situation that you’ve seen before? Is it kind of like the same frequency 4040 10 at all? Not
1:48:06
actually. There’s a there are frequencies. If you think about what happened in the early 1900s There was a epidemic in 1915 there was an epidemic and 17 and then there was a pandemic in 19. So my frequency list from this osteopath includes six frequencies for different kinds of respiratory flu, malignant flu, London flu, there’s six frequencies for flu, and we created a protocol for this respiratory flu pandemic that we have and on long COVID We use those same frequencies in whatever tissue has been affected. So if this patient had COVID, then all of a sudden certain portions of our nervous system stopped working. You could try the virus frequencies in those tissues. So there’s a patient that came in and that has not been able to smell since she had the virus. And I use the virus frequencies on the ethmoid sinus and on the capillaries in the ethmoid sinus and at the end of it, so it took about 3540 minutes at the end of it. She could smell coffee. So the sense of smell came back, so it would be worth a try. And then Deborah Adel frequencies being specifically fucking residence. Yes. So it’s worth a try. I have no idea if it’s gonna help.
1:49:50
Yeah, and I forgot to mention she does have Ehlers Danlos Syndrome. And so I didn’t know if I figured that that went along what you said and it makes so much sense that the the the vagus nerve is getting stretched in and irritated and essentially not functioning as well. Leading to hypo, hypo motility and then in her case, she just lost her ability to control those thinkers. But yeah, so So maybe a combination of treating sorry.
1:50:27
Yeah, it’s just whatever part of the nervous system controls maturation and, and would be parasympathetic, I would think, and to treat the medics and the and the vagus with the virus frequencies and see if it helps, can’t hurt. And you don’t know, work until you use it. So.
1:50:49
Okay. Well,
1:50:53
thank you very much.
Bill Clearfield 1:50:54
There’s two more questions one about does it have any connection with Huntington’s and does it have any does it do any your frequency specific treatments for? Does it help by any issues?
1:51:06
So I haven’t seen Huntington so I don’t know. And then we have one published paper on so actually a controlled trial. On wet and dry or dry and wet macular degeneration. It’s more effective in dry macular degeneration that’s in the published papers section of our website. Rheumatoid arthritis pain Yes. neuropathy. Yes. diabetic neuropathy. Yes, chemotherapy. neuropathies. We do well with Vin Kristen. Not so well with cisplatin or then the platinum chemo agents, rheumatoid arthritis pain, yes, but it’s temporary because you’ve got to turn the vagus back on to control inflammation and most of them are on immune suppressants. So, but peripheral neuropathy is radiculopathy is nerve pain, honestly, is the easiest thing we treat. It’s kind of crazy, but it’s true. And then obviously thalamic pain. That’s pretty amazing. Great.
Bill Clearfield 1:52:18
Thank you so much, Carol. John. You have anything for us. This is my right hand man here. Yeah.
1:52:26
Just a lot. Of
Bill Clearfield 1:52:28
hanging out with guys with beards here.
1:52:32
Parallel just very inspiring. Again, this is so wonderful to hear about this stuff. And especially since you mentioned Flexner and homeopathy, and because I deal with some of the German stuff which is homeopathy. neurotherapy they made their big discoveries in the 30s. So we’re going lash back to but the resistance we’ve had to homeopathy has been extreme in the last few years.
1:53:02
Sure. Now they’re after us. Again. Pharmaceutical companies don’t want to be threatened. Bless their hearts. Well, you guys.
1:53:12
I want to say I want to pick your brain a little bit. They happen to be building an osteopathic school. 30 minutes from my home in West Virginia. They’re very, they’re very open minded. They’re a real real osteopath. Right? They’re walking very, very slowly, very carefully, and keeping their mouth shut about a lot of things but they also are very aware of the homeopathy and are very advanced practitioners, but they can’t talk about it much. So with the new school, starting, they hope to do some research and we hope to work with them and maybe we could work with you with the research and microcurrent as well. Yeah,
1:53:56
the nice thing about FSM is because what I teach is protected by the First Amendment. I talk I’m a clinician sharing like clinical research with other clinicians, and the devices are completely separate company. I’m not a part of that. So the devices are regulated as if they are attends devices. And we just stay away from the devices. I just talked about the effect of the frequencies. So that that’s the way we do it. And I I talk about FSM sometimes as electronic homeopathy. So it’s just simpler because it’s very specific. You know, if you look at a homeopathic remedy, there’s a whole page of indications, right and then it’s doing homeopathic, what do you call it prescription, figuring out what to give somebody? It may take weeks or a month to know, with FSM you know, in 10 to 30 minutes if it’s going to if it’s going to work and how well it’s going to work. So I’m congratulations on the osteopathic College.
1:55:15
Oh, that’s fancy. Everything you’re saying is fantastic. Thank you very much.
Bill Clearfield 1:55:20
Thanks. Again. Thank you so much. I know you’re traveling again, right?
1:55:23
Yeah, tomorrow. We’re headed for Orlando. So great. You guys have a good time. Thanks for having me.
Bill Clearfield 1:55:30
Thank you and we will certainly want you back again. video will be up as soon as I can get it get it up. And the slideshow also, I have here in her in the chat. I have the specific frequency frequency specific.com Does that right?
1:55:50
Yep. Frequency specific.com.
Bill Clearfield 1:55:53
Okay, and then the LSR D that’s our that’s where all our videos and slide notes are. Okay. Dr. Stein put up I don’t know what what article that is. It’s, it’s from from some journal. I’m not sure what it is. But there’s there’s a link to an article there. Next week, Dr. Hal Lawson is going to be telling talking about his latest discovery and so he’s got something interesting to share with us. I will probably not be here. I am going to visit grandbabies on Friday morning and I will be through next Tuesday. So Joel is going to be taking over. Don’t be kind to him. Don’t throw too many tomatoes. I’ll be in Michigan. I’ll try to get on but I don’t know if I’ll be able to or not. And also just look ahead. July 4 is a Tuesday and I’m taking it off. So we’re going to be off that week. So so you’ll just have to, you know, bear with us for one week. And so
1:57:02
can I steal from you Dr. Carol because her presentation was awesome. And phenomena and there’s lots of things that we can add to what she’s she’s presenting. Yep. Okay, okay.
Bill Clearfield 1:57:14
Okay. So any questions, comments, complaints? John got anything else? was good. Okay, all’s good. Joe. Got anything.
1:57:25
Anybody else?
Bill Clearfield 1:57:29
See Dr. Benoit was on this. This this week. We’re going to we’re going to see if we can get him back. Anybody else? Has anybody who has anything that they’d like to present? Please let me know. You know, we’re always open. And next week again, Dr. Helaas, will be speaking for us. And Dr. Cruz. Nice to see you again. And everybody else there were a couple of new names that Dr. Stone a couple of new names, Dr. Smith, and some old friends too. So we’ll be we’ll be out. Same time, same station and we’ll we’ll be seeing you next week. So Goodnight, everybody. Have a great week. Thank you